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eRAM

encyclopedia of Rare Disease Annotation for Precision Medicine




Disease focal segmental glomerulosclerosis
Comorbidity C0033687|proteinuria
Sentences 24
PubMedID- 22584790 Hiv-1 associated nephropathy (hivan) is a clinical and renal histological disease characterized by the presence of heavy proteinuria associated with focal segmental glomerulosclerosis and microcystic tubular dilatation.
PubMedID- 25964888 Four patients in the tbmn group had nephrotic range proteinuria (3 with co-existing fsgs, one with collapsing fsgs).
PubMedID- 24860861 Although “late” recurrence may occur years after transplantation, the most frequent and marked recurrence of fsgs is with massive proteinuria when the graft is reperfused at the time of surgery.
PubMedID- 21030574 Recurrence of focal segmental glomerulosclerosis (fsgs) is often associated with nephrotic proteinuria and is more frequent in children, in patients with rapid progression of the original disease, and in those who lost a previous transplant from recurrence.
PubMedID- 25949442 In this family, both the proband and his father developed glomerular proteinuria with fsgs lesions on renal histology, strongly suggesting that a mutation in one of the genes involved in autosomal dominant fsgs may be the underlying cause.
PubMedID- 22162721 Childhood hiv-1 associated nephropathy (hivan) is a clinical and renal histological disease characterized by heavy proteinuria associated with focal and segmental glomerular sclerosis and/or mesangial hyperplasia in combination with microcystic tubular dilatation.
PubMedID- 21357516 The imai rat is a model of spontaneous focal glomerulosclerosis, which leads to heavy proteinuria, hyperlipidemia, hypertension, and progressive renal failure.
PubMedID- 21034351 Group ii comprised 17 children with persistent proteinuria due to focal segmental glomerulosclerosis (fsgs).
PubMedID- 20127383 Diagnosis of bartter syndrome was made later, as our patient was followed for megacystis and megaureter secondary to the neurogenic bladder and gh deficiency initially; and proteinuria associated with focal segmental glomerulosclerosis responded to treatment for bartter syndrome.
PubMedID- 24069362 focal segmental glomerulosclerosis (fsgs) manifests with heavy proteinuria in association with focal, but progressive, glomerular sclerosis in the kidney [1-3].
PubMedID- 23463342 The level of mir-192 was positively correlated with the proteinuria in patients with fsgs and mcd (r = 0.62, p < 0.001, r = 0.84, p < 0.001, respectively).
PubMedID- 25168833 On the basis of mostly retrospective data, first-line treatment for idiopathic fsgs patients with nephrotic-range proteinuria is a prolonged course of corticosteroids.
PubMedID- 26041001 Recurrence of nephrotic proteinuria in children with focal segmental glomerulosclerosis - early treatment with plasmapheresis and immunoadsorption should be associated with better prognosis.
PubMedID- 25185729 This study aimed to evaluate if circulating supar levels are independently associated with proteinuria in patients with non-fsgs glomerulonephritis.
PubMedID- 25984061 Her ongoing mild proteinuria, hypertension and development of focal sclerosing glomerulonephritis in subsequent kidney biopsy a year later forebode a guarded prognosis.
PubMedID- 22574774 Furthermore, injection of the cd24+cd133+pdx− fraction produced podocytes and tubular cells in mice with adriamycin-induced renal injury (a model of the human podocyte disease known as focal segmental glomerulosclerosis), and was associated with reduced proteinuria [103].
PubMedID- 25654130 Subsequently the concentrating ability is reduced; there is focal segmental glomerulosclerosis with proteinuria, papillary necrosis, and reduced glomerular filtration [53].
PubMedID- 25889917 Adriamycin induces focal segmental glomerulosclerosis with severe proteinuria with interstitial degeneration of the kidney [24].
PubMedID- 19809785 The findings suggest that both hydrocortisone therapy and normalized thyroid hormone status played a pivotal role in the improvement of proteinuria associated with focal segmental glomerulosclerosis.
PubMedID- 26138234 Background/aims: a subset of patients who present with proteinuria and are diagnosed with focal segmental glomerulosclerosis (fsgs) have inherited heterozygous col4a3/a4 mutations and are also diagnosed with thin basement membrane nephropathy (tbmn-omim: 141200).
PubMedID- 22834973 Describes medullary nephrocalcinosis, distal renal tubular acidosis and proteinuria in a patient with focal segmental glomerulosclerosis[9] but there was no polycythaemia.
PubMedID- 26361683 Case report: we report a case of recurrent fsgs with nephrotic-range proteinuria (urine protein creatinine ratio [upcr], >50) and debilitating edema that was resistant to rituximab and plasmapheresis.
PubMedID- 25218681 Last, plasma mir-186, but not mir-125b, level was correlated with degree of proteinuria in patients with fsgs (151 samples).
PubMedID- 22185970 Resolution of proteinuria in a patient with focal segmental glomerulosclerosis following bipap initiation for obesity hypoventilation syndrome.

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