Home Contact Sitemap

eRAM

encyclopedia of Rare Disease Annotation for Precision Medicine




Disease lyme disease
Symptom C0009450|infection
Sentences 19
PubMedID- 22524232 Based on the observations of this study we propose that neurologic injury in the cns during an infection with the lyme disease spirochete b. burgdorferi could be mediated in part by the direct action of the spirochetes on oligodendrocytes or via inflammation mediated by b. burgdorferi in oligodendrocytes.
PubMedID- 23346260 Special attention should be paid to the clinical, serological, and csf findings compatible with b. burgdorferi infection, as successful treatment of early lyme disease can prevent the expensive and devastating consequences of late/chronic lyme disease.
PubMedID- 24009501 Analysis of the transcriptome of borrelia burgdorferi, the causative agent of lyme disease, during infection has proven difficult due to the low spirochete loads in the mammalian tissues.
PubMedID- 25517089 Sensitization resulted in a cytokine skew favoring a th2 bias as well as partial (57%) protection to infection with lyme disease spirochetes (borrelia burgdorferi) following infected tick challenge when compared to tick naive counterparts.
PubMedID- 23042964 Methods: blood culture and serology were used to detect hga infection in patients with early lyme disease who presented with erythema migrans.
PubMedID- 23940747 Murine infection with the lyme disease spirochete borrelia burgdorferi), innate immunity has been shown to be critical for limiting pathogen burdens , , .
PubMedID- 26430734 A widely reported hypothesis for the variation in tick infection rates with the lyme disease pathogen (borrelia burgdorferi) is the “dilution effect”.
PubMedID- 24416278 Although human risk of infection with lyme disease was best predicted by a self-reported previous lyme disease diagnosis, we excluded this variable from further analyses to gain insights into specific behavioral and environmental risk factors.
PubMedID- 22108011 Burgdorferi infection prevalence, the risk of acquiring lyme disease appears to be low.
PubMedID- 23967405 Discovery of the spirochetal aggrecanase raises many questions about the pathogenesis of lyme arthritis and lends support to the concept of persistent b. burgdorferi infection in patients with chronic lyme disease symptoms.
PubMedID- 22294245 Lastly, a predictive relationship between the triggering event of infection with lyme disease and the onset of persistent symptoms cannot be established.
PubMedID- 26469785 In support of the literature , the study suggests a high co-infection rate with either lyme disease or ehrlichiosis for recorded babesiosis cases.
PubMedID- 25976982 (2010) as a co-infection with lyme borreliosis, caused by a parasite with a homology of 98.9 % to b. divergens or b. venatorum.
PubMedID- 23400696 According to the laws of probability, a concurrent hga infection in patients with lyme disease might amount to a few percent at most.
PubMedID- 25870274 Hypercholesterolemia and apoe deficiency result in severe infection with lyme disease and relapsing-fever borrelia.
PubMedID- 22590620 Examining the ability to detect b. burgdorferi infection in patients with early lyme disease at the initial physician visit with either laboratory test, ia/pcr/esi-ms or two-tiered serology, resulted in 19 of 21 patient specimens (90% with a 95% confidence interval of 70% to 99%) having a positive detection of b. burgdorferi infection.
PubMedID- 26370123 Severe symptomatic babesiosis co-infection with lyme disease.
PubMedID- 24950221 Analysis of the transcriptome of borrelia burgdorferi, the causative agent of lyme disease, during infection has proven difficult due to the low spirochete loads in the mammalian tissues.
PubMedID- 25671085 Subsequent investigations revealed evidence of anti- borrelia seroactivity in eia-igg and igg-blot, suggesting a recent infection with lyme disease, and resulted in a 4-week treatment with oral doxycycline.

Page: 1