Disease | duodenal ulcer |
Symptom | C0009450|infection |
Sentences | 17 |
PubMedID- 22829807 | We recently investigated the prevalence of dupa and vir gene homologs and the associations between the status of dupa clusters and clinical outcomes in the us population and found that the presence of a complete dupa cluster increases the duodenal ulcer risk compared with h. pylori infection with incomplete dupa clusters or without the dupa gene independent of the cag pai status (adjusted or, 2.13; 95% ci = 1.13–4.03) . |
PubMedID- 23224792 | Pylori infection in patients with gastroduodenal ulcers. |
PubMedID- 23833377 | the vast majority of primary duodenal ulcers are associated with h. pylori infection of the gastric antral mucosa. |
PubMedID- 22230167 | Helicobacter pylori infection is strongly associated with gastric and duodenal ulcers in a large prospective study. |
PubMedID- 24010097 | Therefore, duodenal ulcers are highly associated with h. pylori infection regardless of prevalence. |
PubMedID- 23158404 | Conclusions: eradication therapy should be provided to patients with h pylori infection after simple closure of perforated gastroduodenal ulcers. |
PubMedID- 22537571 | There is definite role for helicobacter pylori infection in the etiopathogenesis of duodenal ulcer . |
PubMedID- 24187492 | Because reductions in gastric and duodenal barriers often occur in the aging process, elderly patients are at higher risk for hp-negative, nsaid-negative ulcers.1 in a pooled analysis with more than 2,900 patients, 27% of duodenal ulcers were not associated with hp infection or nsaid use.52 in a prospective study of chinese patients with bleeding ulcers in hong kong, the authors reported an increase in the proportion of hp-negative, nsaid-negative idiopathic ulcers from 4.2% in 1997–1998 to 18.8% in 2000.11 worse clinical outcomes have also been reported in patients with hp-negative, nsaid-negative idiopathic ulcers. |
PubMedID- 24598794 | duodenal ulceration was strongly associated with h. pylori infection (or 6.2 ; p<0.001). |
PubMedID- 21687723 | H. pylori is the etiological agent of peptic ulcer disease, 75% of gastric ulcers and 90% of duodenal ulcers are attributed to h. pylori infection (ernst and gold, 2000), as well as two distinct forms of gastric cancer: mucosa-associated lymphoid tissue (malt) lymphoma and adenocarcinoma (parsonnet et al., 1991, 1994; talley et al., 1991; blaser, 1998). |
PubMedID- 22204799 | Effects of melatonin and tryptophan on healing of gastric and duodenal ulcers with helicobacter pylori infection in humans. |
PubMedID- 24834225 | The results of this study supports the results of other studies that suggest the incidence of h.pylori infection related with duodenal ulcer is common, and that non-h pylori and non-nsaids duodenal ulcer is also common. |
PubMedID- 23921679 | Development of an in-house enzyme-linked immunosorbent assay based on surface whole cell antigen for diagnosis of helicobacter pylori infection in patients with gastroduodenal ulcer disease. |
PubMedID- 26516382 | The prevalence of helicobacter pylori infection in patients with gastric and duodenal ulcers - a 10-year, single-centre experience. |
PubMedID- 22352226 | A total of 70 duodenal ulcer patients with helicobacter pylori infection were included in the study. |
PubMedID- 23606834 | Is helicobacter pylori infection the primary cause of duodenal ulceration or a secondary factor. |
PubMedID- 24665577 | Bleeding gastroduodenal ulcers in patients without helicobacter pylori infection and without exposure to non-steroidal anti-inflammatory drugs. |
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