Disease | cryptococcosis |
Symptom | C0876973|pulmonary infection |
Sentences | 9 |
PubMedID- 23532373 | In the murine model of pulmonary infection with cryptococcus neoformans, il-4 receptor alpha (il-4ralpha)-dependent polyfunctional t(h)2 cells induce disease progression associated with alternative activation of lung macrophages. |
PubMedID- 21383052 | The immune response to cryptococcus neoformans following pulmonary infection of c57bl/6 wild-type (wt) mice results in the development of persistent infection with characteristics of allergic bronchopulmonary mycosis (abpm). |
PubMedID- 20864680 | Persistent pulmonary infection with cryptococcus neoformans in c57bl/6 mice results in chronic inflammation that is characterized by an injurious th2 immune response. |
PubMedID- 24470469 | Defect of card9 leads to impaired accumulation of gamma interferon-producing memory phenotype t cells in lungs and increased susceptibility to pulmonary infection with cryptococcus neoformans. |
PubMedID- 25422263 | Dectin-2 deficiency promotes th2 response and mucin production in the lungs after pulmonary infection with cryptococcus neoformans. |
PubMedID- 21097509 | Gpr34-deficient mice showed significantly increased paw swelling in the delayed type hypersensitivity test and higher pathogen burden in extrapulmonary tissues after pulmonary infection with cryptococcus neoformans compared with wild-type mice. |
PubMedID- 20056835 | pulmonary infection with an interferon-gamma-producing cryptococcus neoformans strain results in classical macrophage activation and protection. |
PubMedID- 25013584 | Concomitant pulmonary infections with cryptococcus neoformans and burkholderia cepacia in lung transplant recipients are very rare and create unique diagnostic and therapeutic dilemmas. |
PubMedID- 23175699 | We quantified and determined the molecular derivation of b-1a, b-1b, and b-2 b cell populations in c57bl/6 mice after pulmonary infection with cryptococcus neoformans. |
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