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PedAM

Pediatric Disease Annotations & Medicines




Disease sudden cardiac death
Phenotype C0018801|heart failure
Sentences 53
PubMedID- 20109482 The mechanisms responsible for sudden cardiac death in heart failure (hf) are unclear.
PubMedID- 23111739 An increase in qt dispersion is a possible substrate for ventricular arrhythmias and sudden cardiac death in patients with chronic heart failure [12], left ventricular hypertrophy [22], diabetes mellitus [23], prolonged qt interval [24], obesity [25] and in subjects who are 55 years of age or older [18].
PubMedID- 24418727 A common nos1ap genetic polymorphism, rs12567209 g>a, is associated with sudden cardiac death in patients with chronic heart failure in the chinese han population.
PubMedID- 23848972 Beta-blockers for the prevention of sudden cardiac death in heart failure patients: a meta-analysis of randomized controlled trials.
PubMedID- 25302657 A prediction model for sudden cardiac death in patients with heart failure and preserved ejection fraction.
PubMedID- 26483614 Objective: heart rate variability (hrv) has been reported to be an independent predictor of all-cause and sudden cardiac death in patients with heart failure.
PubMedID- 21682151 On the other hand, there was much debate on whether these results could be extrapolated for patients with non-ischemic cardiomyopathy until the sudden cardiac death in heart failure trial (scd-heft) demonstrated a significant benefit of this therapy.
PubMedID- 24556243 Elevation of inflammatory markers is associated with sudden cardiac death in patients with heart failure or coronary artery disease, and onset of ventricular arrhythmia.32–35 of note, there is abundant evidence that elevated serum levels of crp are associated with the genesis and perpetuation of af.
PubMedID- PMC4328619 However, the majority of sudden cardiac death (scd) in patients with heart failure occurs in those with mild-moderate systolic heart failure (lvef 36%-50%) who currently do not qualify for an icd.
PubMedID- 23967088 Hypertrophic cardiomyopathy (hcm) is an autosomal dominant disorder of the myocardium which is hypertrophied resulting in arrhythmias and heart failure leading to sudden cardiac death (scd).
PubMedID- 20938735 Abnormal sympathetic innervation and activation, as assessed with 123-i mibg is predictive of cardiac mortality, morbidity, and ventricular arrhythmias or sudden cardiac death in patients with heart failure.
PubMedID- 21384153 Subsequent trials, multicenter automatic defibrillator implantation trial ii (madit-ii) and sudden cardiac death in heart failure trial (scd-heft), used lvef without additional high-risk markers for ventricular arrhythmia.
PubMedID- 21439494 Epidemiology of sudden cardiac death in patients with heart failure.
PubMedID- 23397903 Early repolarization pattern associated with sudden cardiac death: long-term follow-up in patients with chronic heart failure.
PubMedID- 25698991 sudden cardiac death in heart failure/psychosocial factors outcome study in sudden cardiac death.
PubMedID- 23070331 Implantable cardioverter defibrillators (icd) are used as standard therapy to prevent sudden cardiac death in heart failure patients.
PubMedID- 22830024 In the adult population, dcm is the most common form of non-ischemic cardiomyopathy and a major cause of the heart failure leading to sudden cardiac death.
PubMedID- 23568645 Asian sudden cardiac death in heart failure (asian-hf) registry.
PubMedID- 21960418 The sudden cardiac death in heart failure trial (scd-heft) showed 23% reduction in relative risk of mortality in patients with lvef ≤35% and new york heart association (nyha) class ii–iii congestive heart failure due to ischemic or non-ischemic etiology [2].
PubMedID- 22806059 Another point of interest is the role of 123i-mibg in predicting sudden cardiac death in patients with heart failure and even the predictive value of appropriate implantable cardioverter-defibrillator (icd) therapy.
PubMedID- 22713286 The predictive values of beta1-adrenergic and m2 muscarinic receptor autoantibodies for sudden cardiac death in patients with chronic heart failure.
PubMedID- 22968848 Aims: this cross-sectional study evaluated the application of accepted international implantable cardioverter defibrillator (icd) guidelines for primary prevention of sudden cardiac death in patients with heart failure.
PubMedID- 23047575 Abnormal myocardial i-123-mibg uptake can also be indicative of higher risk of ventricular arrhythmias and sudden cardiac death inpatients with heart failure.
PubMedID- 24475137 Previous studies have shown that spatial repolarization heterogeneity is linked to arrhythmia and sudden cardiac death in patients with myocardial infarction, heart failure, and long qt syndrome [46]–[49].
PubMedID- 26251283 Although implantable cardioverter-defibrillators (icds) prevent sudden cardiac death in patients with heart failure (hf) and reduced ejection fraction,1–4 it is not clear if these devices are associated with improved outcomes among older patients with hf, especially those with significant comorbidities or multiple hospitalizations for hf.
PubMedID- 20440250 These repolarization gradients form an electrophysiological substrate that predisposes to arrhythmias and sudden cardiac death in heart failure.
PubMedID- 25464420 Serial (1)(2)(3)i-metaiodobenzylguanidine imaging predicts the risk of sudden cardiac death in patients with chronic heart failure.
PubMedID- 26194143 Background: ventricular arrhythmia is the major cause of sudden cardiac death for patients with heart failure, including those receiving implantation of cardiac resynchronization therapy (crt).
PubMedID- 22024149 Average t-wave alternans activity in ambulatory ecg records predicts sudden cardiac death in patients with chronic heart failure.
PubMedID- 25135287 In the cardiac insufficiency bisoprolol study ii (cibis-ii), bisoprolol reduced all-cause mortality by 34% and sudden cardiac death by 44% in patients with heart failure [30].
PubMedID- 25138427 Ventricular tachycardia (vt) is a major cause of sudden cardiac death (scd) in patients with heart failure (hf).
PubMedID- 24286584 sudden cardiac death in heart failure.
PubMedID- 26044253 This important neurovisceral/autonomic nervous system also plays a major role in the pathophysiology and progression of heart disease, including heart failure and arrhythmias leading to sudden cardiac death.
PubMedID- 20662865 Recent studies report surprisingly low rates of implantable cardioverter defibrillator (icd) placement for primary prevention against sudden cardiac death among patients with heart failure and left ventricular systolic dysfunction.
PubMedID- 22176998 A total of 245 patients (49%) satisfied the "sudden cardiac death in heart failure trial (scd-heft) criteria," defined as a left ventricular ejection fraction of
PubMedID- 21609390 Prediction values of t wave alternans for sudden cardiac death in patients with chronic heart failure: a brief review.
PubMedID- 21704222 Background and purpose: left ventricular ejection fraction lacks specificity to predict sudden cardiac death in heart failure.
PubMedID- 24285944 The sudden cardiac death in heart failure trial (scd-heft)[8] showed that hf patients with icd therapy had an all-cause death risk of 23% lower than placebo and an absolute decrease in mortality of 7.2% after 5-year follow-up in the overall population.
PubMedID- 24812112 For example, the sudden cardiac death in heart failure trial (scd-heft) found that the mortality benefit started to appear approximately 1.5 years after implantation of an icd.1 all patients were followed until the occurrence of the outcomes and death from the index date, which was the date of implantation for those who received icd therapy and the discharge date from their admission to hospital for heart failure for those who did not receive icd therapy.
PubMedID- 23662051 Background and purpose: qt and t(peak)-t(end) (te) intervals are associated with sudden cardiac death in patients with chronic heart failure (chf).
PubMedID- 23040109 sudden cardiac death in heart failure patients with preserved ejection fraction.
PubMedID- 21439495 sudden cardiac death risk stratification in patients with heart failure.
PubMedID- 22133464 Decreased scale-specific heart rate variability after multiresolution wavelet analysis predicts sudden cardiac death in heart failure patients.
PubMedID- 26097719 However, given the data from the sudden cardiac death in heart failure trial (scd-heft) [5], the percentage of primary prevention patients receiving icd therapy was 21% in 3 years.
PubMedID- 19193457 Heart rate recovery predicts sudden cardiac death in heart failure.
PubMedID- 23781262 The risk of sudden cardiac death in heart failure patients is six to nine times that of the general population, and approximately half of these patients die of ventricular arrhythmias.
PubMedID- 26175568 Analysis of the scd-heft (sudden cardiac death in heart failure trial), which evaluated the benefit of icd treatment in the primary prevention of scd in patients with icm or nicm, an lvef ≤ 35%, and nyha ii or iii functional class, showed that although there is a mortality risk reduction, only about 20-25% of patients received an appropriate icd shock in 5 years (16).
PubMedID- 24727258 (sudden cardiac death in heart failure trial [scd-heft]; nct00000609).
PubMedID- 22503242 These observations suggest a difference between primary heart failure in sudden cardiac death and terminal cardiac dysfunction secondary to fatal asphyxiation or drowning.
PubMedID- 23220154 heart failure (hf) is associated with susceptibility to sudden cardiac death.

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