PedAM
Pediatric Disease Annotations & Medicines
| Disease | pulmonary fibrosis |
| Phenotype | C1145670|respiratory failure |
| Sentences | 14 |
| PubMedID- 25366193 | The clinical course of apap is variable; in severe cases, patients develop lethal respiratory failure due to pulmonary fibrosis. |
| PubMedID- 23272154 | Paraquat poisoning is characterized by multi-organ failure and pulmonary fibrosis leading to respiratory failure, with acute kidney injury being the major cause of mortality [10]. |
| PubMedID- 23968871 | Conclusions: we confirm that use of invasive mv for acute respiratory failure in patients with idiopathic pulmonary fibrosis or fibrosing idiopathic nonspecific interstitial pneumonia is associated with a high mortality; however, a subset of patients may be discharged alive from the intensive care unit and hospital, providing an opportunity to consider lung transplant in case of eligibility. |
| PubMedID- 26000190 | Surfactant protein c deficiency varies from mild temporary tachypnea to lethal respiratory failure due to idiopathic pulmonary fibrosis [11]. |
| PubMedID- 20852840 | Prognostic impact of high-flow nasal cannula oxygen supply in an icu patient with pulmonary fibrosis complicated by acute respiratory failure. |
| PubMedID- 26314316 | The irreversible and rapid progression of pulmonary fibrosis associated with respiratory failure is the main cause of death in the later stages of poisoning. |
| PubMedID- 25201432 | Noninvasive ventilation in the event of acute respiratory failure in patients with idiopathic pulmonary fibrosis: waiting for. |
| PubMedID- 24768565 | Noninvasive ventilation in the event of acute respiratory failure in patients with idiopathic pulmonary fibrosis. |
| PubMedID- 24010554 | Case presentation: we report on a case of accidental paraquat poisoning in a 23 years old caucasian man, who developed respiratory failure due to pulmonary fibrosis. |
| PubMedID- 26568853 | The clinical manifestation of paraquat poisoning can be categorized into three groups: 1) mild poisoning (< 20 mg paraquat ion per kilogram of body weight), in which the patients often have minor gastrointestinal symptoms and usually recover fully; 2) severe poisoning (20 - 40 mg paraquat ion per kilogram of body weight), in which patients develop acute renal failure, acute lung injury and progressive pulmonary fibrosis, which leads to respiratory failure 2 - 3 weeks after the ingestion of paraquat; 3) fulminant poisoning (> 40 mg paraquat ion per kilogram of body weight); this high concentration of paraquat establishes multiple organ failure, leading to death within hours to a few days after the ingestion (7). |
| PubMedID- 21147624 | Patients presented end-stage respiratory failure due to idiopathic pulmonary fibrosis (ipf) (n = 12), other than ipf forms of idiopathic interstitial pneumonia (iip) (n = 15), copd (n = 16), and sarcoidosis (n = 5). |
| PubMedID- 21253589 | Idiopathic pulmonary fibrosis (ipf) can lead to respiratory failure and death due to deteriorating respiratory function [1], [4]. |
| PubMedID- 23605508 | Clinical, radiologic, and laboratory findings suggested a diagnosis of chronic hypoxemic, type 1 respiratory failure, due to combined pulmonary fibrosis and emphysema, complicated by severe, precapillary pulmonary hypertension. |
| PubMedID- 23772158 | It is characterized by nonproductive cough and dyspnea followed by rapidly progressive respiratory failure with pulmonary fibrosis showing with high mortality. |
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