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PedAM

Pediatric Disease Annotations & Medicines




Disease proteinuria
Phenotype C0035078|renal failure
Sentences 30
PubMedID- 24550645 Clinical manifestations such as skin, joint, gastro-intestinal, and renal involvement and outcomes such as chronic renal failure, persistence of hematuria, or proteinuria were assessed.
PubMedID- 26487672 Conclusions: lact supplementation mitigated the systemic inflammation and proteinuria associated with renal failure, suggesting that in the gut microbiota, lact plays a protective role against the progression of ckd.
PubMedID- 20813071 The most common clinical manifestation of amyloidosis is the development of proteinuria that may lead to renal failure.
PubMedID- 24843463 Enrolled patients did not have hepatic insufficiency (alanine transaminase or aspirate transaminase is ≥2‐fold of the upper reference limit of each institute), renal insufficiency (serum creatinine ≥1.4 mg/dl), severe diabetic complications (overt proteinuria with renal failure, unstable proliferative retinopathy or symptomatic orthostatic hypotension), malignant tumors or dementia.
PubMedID- 24555136 We present a patient who developed renal al kappa amyloidosis manifested by progressive renal failure without proteinuria after several years of mgus with stable paraprotein levels and renal function.
PubMedID- 22025969 Human diabetic nephropathy is characterized by changes in gfr with increasing proteinuria which leads to renal failure [2, 3].
PubMedID- 21524952 renal failure can be associated with moderate proteinuria, without hematuria.
PubMedID- 23802112 Microhematuria and proteinuria in association with acute renal failure developed during the course of the disease.
PubMedID- 24533557 At the age of 51 years, rapidly progressive renal failure occurred with nephrotic proteinuria, and ncgn with subepithelial deposits was revealed by renal biopsy.
PubMedID- 21782136 The degree of proteinuria associated with renal failure is a generally well accepted marker of disease severity.
PubMedID- 20591157 However, our patient was diagnosed as chronic renal failure with proteinuria and hypoalbuminemia (table 1).
PubMedID- 21393360 Background: chronic renal failure (crf) is associated with hypertension, proteinuria, loss of myogenic constriction (mc) of mesenteric arteries and increased production of reactive oxygen species (ros) under experimental conditions.
PubMedID- 22552385 Both developed proteinuria associated with acute renal failure in early childhood.
PubMedID- 23889996 Renal involvement may manifest as hematuria and proteinuria and can lead to renal failure.
PubMedID- 22888268 Conclusion: podocyte exposure to hypoxia leads to reduced viability and sd protein expression, which may explain persistent and/or increasing proteinuria in patients with progressive renal failure.
PubMedID- 24293658 The diagnosis of hus was focused on the association between thrombocytopenia (platelets <150 g/l), mechanical haemolysis (anaemia, increase in lactated dehydrogenase (ldh) serum levels, undetectable serum haptoglobin and schizocytes when present) and acute kidney injury (aki; proteinuria and haematuria with or without renal failure) [13, 25, 29].
PubMedID- 20671931 Renal biopsy is the gold standard for the diagnosis and follow-up, whereas the measurement of proteinuria identifies patients with overt renal failure, but fails to detect early silent disease.
PubMedID- 22166090 The most common manifestation of wg in the kidney is segmental necrotizing glomerulonephritis with proteinuria that often leads to rapidly progressive renal failure [1].
PubMedID- 26351598 Our hypothesis may be supported by the fact that two doses of pulse methylprednisolone achieved only partial remission and a subsequent course of oral prednisone did not prevent the development of acute renal failure with persistently increased proteinuria and serum creatinine levels.
PubMedID- 21372294 renal failure attributable to proteinuria and uremia also induces er stress within the kidney, which contributes to the transformation of tubular epithelial cells to a fibroblast-like phenotype, fibrosis, and tubular cell apoptosis, further diminishing renal function.
PubMedID- 24167517 After 3 weeks the patient was admitted to our unit for acute renal failure with mild proteinuria with negligible urinary sediment.
PubMedID- 23738826 In our case report, two out of five have proteinuria without renal failure and not associated with nephrocalcinosis.
PubMedID- 22807634 While the development of advanced diabetic nephropathy is marked clinically by proteinuria which subsequently leads to renal failure, it had been reported that low glomerular filtration rate is already present in a substantial proportion of type 2 diabetic patients even though they were microalbuminuric or even normoalbuminuric.1–4 fifty-one percent of patients in the uk prospective diabetes study who developed chronic renal failure remained nonalbuminuric.2 the prevalence of low estimated glomerular filtration rate (egfr) in normoalbuminuric type 2 diabetic patients living in brazil and australia was 12.7% and 23%, respectively.1,3 low egfr was present in 12.6% of east asian normoalbuminuric type 2 diabetic patients in korea.5 in nonproteinuric type 2 diabetic japanese and chinese patients, the prevalence of low renal function had been reported as 11.4%6 and 19.7%,7 respectively.
PubMedID- 26123310 A recent development of mild renal failure with moderate proteinuria led us to perform a kidney biopsy.
PubMedID- 26035383 Anti-proteinuric interventions are not always successful, and residual proteinuria often leads to renal failure.
PubMedID- 24335780 Hfrs is characterized by acute renal failure with often massive proteinuria caused by tubular and glomerular involvement [6].
PubMedID- 21599942 Renal complications start with silent glomerular hyperfiltration before the development of microalbuminuria then proteinuria, which can lead to renal failure [2].
PubMedID- 21525478 Terminal renal failure without major proteinuria and with small kidneys (presumably ischemic nephropathy) was seen in 11% of the patients, and diabetes in the presence of known primary kidney disease was seen in 19% of the patients.
PubMedID- 22991235 Moreover, in wolcott–rallison syndrome, renal dysfunction, with persistent proteinuria leading to renal failure, was reported in 45% of the patients [ozbek et al., 2010].
PubMedID- 23427756 Introduction: we report a case of refractory hypertension and acute renal failure with mild proteinuria due to an unreported bilateral renal artery stenosis, who underwent renal biopsy in the suspicion of rapidly progressive glomerulonephritis.

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