Disease | multiple sclerosis |
Phenotype | C0014038|encephalitis |
Sentences | 8 |
PubMedID- 24876950 | Moreover, in experimental autoimmune encephalitis, a model of multiple sclerosis, it has been observed that il-17 is able to breach tight junction of the blood-brain barrier in vivo [92]. |
PubMedID- 22708060 | In the experimental autoimmune encephalitis (eae) model, ananalogue of multiple sclerosis in mice, systemic transplantation of mscs to theaffected mice prevented the development of inflammatory infiltrates (t and b cells,macrophages) and that of the demyelination process in the cns; moreover, it reducedthe response of t cells to mog peptides which originate from myelin [91]. |
PubMedID- 25877850 | This report presents the 4th documented case worldwide of herpes simplex encephalitis in multiple sclerosis (ms) patients treated with natalizumab and the first case in the uk. |
PubMedID- 22048950 | In this case report we describe the first non-fatal herpes simplex virus encephalitis (hse) case with natalizumab for multiple sclerosis (ms). |
PubMedID- 24303066 | T cell activation plays an important role in inflammation-related neuronal injury associated with diseases including encephalitis, the progressive forms of multiple sclerosis [1–3] and a wide variety of other neuroinflammatory diseases. |
PubMedID- 25890218 | Similarly, monocyte infiltration correlated with progression to the paralytic stage in experimental allergic encephalitis, the mouse model of multiple sclerosis, whereas inhibiting monocyte recruitment to the cns blocked disease progression [49]. |
PubMedID- 22152786 | Anti-n-methyl d-aspartate-type glutamate receptor antibody-positive limbic encephalitis in a patient with multiple sclerosis. |
PubMedID- 25876479 | A case of anti-n-methyl-d-aspartate receptor encephalitis with multiple sclerosis-like demyelinated lesions. |
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