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PedAM

Pediatric Disease Annotations & Medicines




Disease malnutrition
Phenotype C0023890|liver cirrhosis
Sentences 13
PubMedID- 20576106 Causes for malnutrition in liver cirrhosis are known to include a reduction in oral intake (for various causes), increased protein catabolism and insufficient synthesis, and malabsorption/maldigestion associated with portal hypertension [3,5,6].
PubMedID- 20618455 Aim: a late evening snack (les) is recommended for protein-energy malnutrition in patients with liver cirrhosis.
PubMedID- 26036456 The patient had a past history of liver cirrhosis hcv-related with a severe malnutrition, hypertrophic cardiomyopathy, diverticular disease, hiatal ernia, previous appendectomy.
PubMedID- 23936183 Bcaa has been used as a supplemental therapy to improve malnutrition in patients with liver cirrhosis [1].
PubMedID- 24829672 malnutrition in liver cirrhosis:the influence of protein and sodium.
PubMedID- 23027617 Branched-chain amino acids (bcaa), which improve protein malnutrition in patients with liver cirrhosis, reduce the risk of hepatocellular carcinoma in these patients with obesity.
PubMedID- 23601060 Free fatty acid as a marker of energy malnutrition in liver cirrhosis.
PubMedID- 20712878 Bia has been used for the assessment of malnutrition in patients with liver cirrhosis.
PubMedID- 21518402 Conclusion: les with bcaa-enriched nutrient therapy can improve protein malnutrition in patients with liver cirrhosis, and is more useful in the early stages of liver cirrhosis in improving hepatic parenchymal cell mass.
PubMedID- 21422706 Energy malnutrition worsens survival in patients with liver cirrhosis, and is currently defined as non-protein respiratory quotient (nprq) <0.85, as measured by indirect calorimetry.
PubMedID- 22969221 The effect of malnutrition on survival in patients with decompensated liver cirrhosis has not been well defined.
PubMedID- 25789501 We found that supplementation with branched-chain amino acids (bcaa), which improve protein malnutrition in patients with liver cirrhosis [38], significantly inhibited liver carcinogen diethylnitrosamine (den)-induced hepatocarcinogenesis as well as spontaneously occurring hepatic preneoplastic lesions in db/db obese and diabetic mice [34,39].
PubMedID- 24273224 Supplementation with branched-chain amino acids (bcaa), which are used to improve protein malnutrition in patients with liver cirrhosis, can also reduce the risk of hcc in obese cirrhotic patients.

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