PedAM
Pediatric Disease Annotations & Medicines
| Disease | ischemia |
| Phenotype | C0028790|cerebral artery occlusion |
| Sentences | 9 |
| PubMedID- 21611146 | The effects of inhibition of p2 receptors by pyridoxalphosphate-6-azophenyl-2′,4′-disulphonic acid (ppads) on electrophysiological, functional and morphological alterations in an ischemia model with permanent middle cerebral artery occlusion (mcao) were investigated up to day 28. spontaneously hypertensive rats received ppads or vehicle intracerebroventricularly 15 minutes prior mcao for up to 7 days. |
| PubMedID- 25206589 | The middle cerebral artery occlusion model of cerebral ischemia is thought to closely mimic the events of an embolic stroke. |
| PubMedID- 22876219 | In a model of striatal ischemia with endothelin-induced middle cerebral artery occlusion, neural differentiation of transplanted nscs was shown for up to 12 weeks after grafting (buhnemann et al., 2006). |
| PubMedID- 24782904 | The middle cerebral artery occlusion model of focal cerebral ischemia in rat was performed by using intraluminal suture method. |
| PubMedID- 24885160 | Methods: we used a middle cerebral artery occlusion (mcao) model of cerebral ischemia, associated with treatment with tpa, in male spontaneously hypertensive rats (shr). |
| PubMedID- 20109493 | Middle cerebral artery occlusion (mcao), which leads to focal cerebral ischemia, serves as an experimental model for brain stroke. |
| PubMedID- 23050647 | Dpi-induced gene changes were analyzed by microarray analysis from rat brains subjected to 90 min of middle cerebral artery occlusion, treated with nacl (ischemia), dimethylsulfoxide (dmso), or dmso and dpi (dpi), and reperfused for 48 h. |
| PubMedID- 24116277 | In addition, these extracts were also effective in the decrease of infarction volume observed in an in vivo mouse model of ischemia with transient middle cerebral artery occlusion (kang et al., 2006). |
| PubMedID- 21427729 | First, when subjected to a middle cerebral artery occlusion model of focal cerebral ischemia, transgenic fb-adk-def mice, which have increased adk expression in striatum (164%) and reduced adk expression in cortical forebrain (65%), demonstrate increased striatal infarct volume (126%) but almost complete protection of cortex (27%) compared with wild-type (wt) controls, indicating that cerebral injury levels directly correlate to levels of adk in the cns. |
Page: 1