Disease | ischemia |
Phenotype | C0007785|cerebral infarct |
Sentences | 16 |
PubMedID- 24501870 | [a clinical case of young, oral combined contraceptive using women, heterozygous carrier of the factor v (leiden) which revealed thrombosis of the left internal jugular vein and brain ischemia with cerebral infarction and ischemic stroke]. |
PubMedID- 20700423 | Antihypertensive therapy to treat these diseases may exacerbate ischemia in patients with concurrent cerebral infarction. |
PubMedID- 21697985 | The main problem of temporary clipping is the occurrence of brain ischemia, with postoperative cerebral infarction, due to extended time of temporary arterial occlusion. |
PubMedID- 26462796 | Delayed cerebral ischemia (dci) with or without cerebral infarction occurs in about 30 % of patients with asah and is strongly linked to cerebral vasospasm [5, 6]. |
PubMedID- 25823006 | The patients were classified as having clinical evidence of brain embolization if they had either new onset of a persistent focal neurological deficit or a transient ischemic attack (tia) defined as brief episodes of neurological dysfunction resulting from focal cerebral ischemia not associated with permanent cerebral infarction. |
PubMedID- 22645702 | Transient ischemic attack (tia) is a brief episode of neurological dysfunction resulting from focal cerebral ischemia not associated with permanent cerebral infarction [13]. |
PubMedID- 21319356 | Neurologic complications included cerebral infarction, paraplegia due to spinal cord ischemia, and peripheral neuropathy. |
PubMedID- 22567018 | Transient ischemic attacks (tias) are usually brief neurological deficits resulting from focal cerebral ischemia not associated with permanent cerebral infarction [1]. |
PubMedID- 23860031 | This article reviews recent findings on mirna profile changes in neural progenitor cells after cerebral infarction and the contributions of mirnas to their ischemia-induced proliferation and differentiation. |
PubMedID- 23737846 | At 24 h after reperfusion, the remaining rats showed serious neurological deficits (grade 6.5 ± 0.5, n = 19, p > 0.05 versus ischemia) with the cerebral infarct volume approximately one-third of the whole brain (33.4% ± 6.3%, n = 12, p > 0.05 versus ischemia) (table 1 and figure 1(d)). |
PubMedID- 26525737 | All cases of new cerebral ischemia occurred in patients with previous chronic cerebral infarction and were also on the same side as the infarction. |
PubMedID- 20830172 | These results mean that the oct technique has clinical potential for applications to monitor or diagnose a focal degraded area, such as cerebral infarctions due to focal ischemia in brains. |
PubMedID- 20628399 | Moreover, in sah patients who experience overt delayed cerebral ischemia (dci) leading to cerebral infarction, the extent of bbb dysfunction within the infarct would be expected to be similar to that in primary acute stroke affecting the same vascular distribution. |
PubMedID- 24618563 | ischemia-induced cerebral infarction is more severe in older animals as compared to younger animals, and is associated with reduced availability of insulin-like growth factor (igf)-1. |
PubMedID- 24926332 | cerebral infarction may lead to cerebral ischemia, hypoxia, necrosis and finally neurological deficit (1). |
PubMedID- 24098716 | Dci-ci: delayed cerebral ischemia with cerebral infarction. |
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