Disease | interstitial nephritis |
Phenotype | C0033687|proteinuria |
Sentences | 4 |
PubMedID- 26266393 | The most common renal manifestation of igg4-related disease is tubulointerstitial nephritis marked with proteinuria, hematuria, decreased kidney function, hypocomplementemia, and radiologic abnormalities. |
PubMedID- 25880729 | Besides producing a reversible renal failure, nsaids are known to cause acute interstitial nephritis (ain) with hematuria, proteinuria and flank pain [14] as well as acute tubular necrosis (atn). |
PubMedID- 22382248 | interstitial nephritis with proteinuria >1 g/day is uncommon and almost always the result of drug-induced atin with an associated minimal change glomerulonephritis (gn). |
PubMedID- 25684013 | For example, drug exposure can result in the release of permselectivity promoting factors from activated inflammatory or immune cells, causing mcd.13 another possible mechanism of mcd is via direct toxin effects on glomerular epithelial cells, resulting in abnormal permeability and effacement of the foot processes.13 neugarten, et al.7 reported the first case of rifampicin-induced nephrotic syndrome, and described the patient as having acute interstitial nephritis with heavy proteinuria and effacement of the glomerular epithelial cells. |
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