Home Contact Sitemap

PedAM

Pediatric Disease Annotations & Medicines




Disease hyperglycemia
Phenotype C0011854|type 1 diabetes
Sentences 45
PubMedID- 24319123 These results suggest vulnerability of the developing brain in young children to effects of type 1 diabetes associated with chronic hyperglycemia and glucose variability.
PubMedID- 21193630 Unexplained hyperglycemia in a patient with type 1 diabetes may suggest impending dka, and monitoring of ketones should therefore be performed.
PubMedID- 22127749 Defects in insulin action were induced only after 24 hours of hyperglycemia in patients with type 1 diabetes.23 insulin treatment for 4 weeks and good glycemic control improved insulin resistance in patients with uncontrolled type 2 diabetes.24 those studies indicated that hyperglycemia worsens insulin resistance, and that this augmented resistance could be recovered after glycemic control was improved.
PubMedID- 23193216 Pizza is widely recognized to cause marked late postprandial hyperglycemia in patients with type 1 diabetes (11).
PubMedID- 24607838 Acute dehydroepiandrosterone (dhea) administration improves hyperglycemia in rats with streptozotocin (stz)-induced type 1 diabetes mellitus.
PubMedID- 21273491 To our knowledge, no study has yet assessed a possible relationship between the occurrence of ams and hyperglycemia in type 1 diabetes.
PubMedID- 24089261 The effect of sex on endothelial function responses to clamped hyperglycemia in type 1 diabetes.
PubMedID- 25560829 hyperglycemia associated with type 1 diabetes is a consequence of immune-mediated destruction of insulin producing pancreatic beta-cells.
PubMedID- 21071702 Methods and results: chronic hyperglycemia with insulin deficiency (type 1 diabetes) was induced in rats by streptozotocin.
PubMedID- 21059246 It appears that this study showed some promise for control of post prandial hyperglycemia in type 1 diabetes patients [30].
PubMedID- 23250800 Our aim was to determine if increased nitric oxide generation by l-arginine infusion would reverse this deleterious response to clamped hyperglycemia in women with type 1 diabetes mellitus.
PubMedID- 24265381 Mean weights of the various foods were as follows: low-fat dinner, chicken 98 g, rice 160 g, broccoli 96 g, grapes 127 g; high-fat dinner, cheese 74 g, chicken 39 g, white bread 63 g, croutons 32 g, oranges 216 g. it is noteworthy that a recent study (3) demonstrated that higher-fat meals containing 35 g of fat (a quantity intermediate between that given in wolever and mullan [4] and our [2] study) cause late postprandial hyperglycemia in children with type 1 diabetes.
PubMedID- 22701470 Acute reduction of hyperglycemia in type 1 diabetes partially restores but does not normalize endothelial function [15].
PubMedID- 20332358 Conclusions: adjunctive exenatide therapy reduces postprandial hyperglycemia in adolescents with type 1 diabetes.
PubMedID- 20978101 Noninvasive assessment of exercise-related intramyocellular acetylcarnitine in euglycemia and hyperglycemia in patients with type 1 diabetes using (1)h magnetic resonance spectroscopy: a randomized single-blind crossover study.
PubMedID- 21519350 The results showed that tcv treatment decreased hyperglycemia in type 1 diabetes (t1d) induced by multiple low-dose streptozotocin (mld-stz) as compared with the controls, preserved the number of healthy pancreatic islets and increased the production of insulin in the islets.
PubMedID- 22548951 Objective: to observe the effect of the dawn phenomenon on basal glucose and postbreakfast hyperglycemia in patients with type 1 diabetes treated with once-nightly insulin glargine and premeal insulin lispro.
PubMedID- 25948477 Data from the diabetes control and complications trial (dcct) demonstrated that strict control of hyperglycemia in type 1 diabetes patients without clinical neuropathy decreased dpn development in 60% of cases over 5 years of follow-up study16,18.
PubMedID- 26126619 In contrast to injection of stz to adult rats in a lower dose (60 mg/kg) that leads to type 1 diabetes with severe hyperglycemia [14, 24–27], administration of stz to neonatal rats in a higher dose (90–100 mg/kg) leads to acute hyperglycemia within the first few days without resulting in complete loss of insulin production [14, 24–27].
PubMedID- 20841614 Less is known about the renal excretion of inflammatory biomarkers in response to acute clamped hyperglycemia in humans with uncomplicated type 1 diabetes (15).
PubMedID- 20042777 Unexplained hyperglycemia in a patient with type 1 diabetes may suggest impending dka, and monitoring of ketones should therefore be performed.
PubMedID- 25799386 Nmda receptor plasticity might therefore represent a homeostatic compensatory response to chronic hyperglycemia associated with type 1 diabetes.
PubMedID- 23613558 (15) demonstrates the feasibility of allogeneic transplantation of embryonic pancreatic tissue for the correction of hyperglycemia in type 1 diabetes.
PubMedID- 22580727 The pathogenesis of nafld remains obscure, but it has been hypothesized that hepatic fat accumulation in type 1 diabetes may be due to lipoprotein abnormalities, hyperglycemia-induced activation of the transcription factors carbohydrate response element-binding protein (chrebp) and sterol regulatory element-binding protein 1c (srebp-1c), upregulation of glucose transporter 2 (glut2) with subsequent intrahepatic fat synthesis, or a combination of these mechanisms.
PubMedID- 21193625 As retinopathy is estimated to take at least 5 years to develop after the onset of hyperglycemia, patients with type 1 diabetes should have an initial dilated and comprehensive eye examination within 5 years after the onset of diabetes.
PubMedID- 23264428 Unexplained hyperglycemia in a patient with type 1 diabetes may suggest impending dka, and monitoring of ketones should therefore be performed.
PubMedID- 23264422 As retinopathy is estimated to take at least 5 years to develop after the onset of hyperglycemia, patients with type 1 diabetes should have an initial dilated and comprehensive eye examination within 5 years after the onset of diabetes.
PubMedID- 21964158 Naringin ameliorates atherogenic dyslipidemia but not hyperglycemia in rats with type 1 diabetes.
PubMedID- 20299466 (13) provided evidence that posthypoglycemic hyperglycemia in patients with type 1 diabetes is the result of counterregulatory hormonal response to hypoglycemia in concert with prevalent plasma insulin concentration, and that all of the hormones but glucagon may play a role.
PubMedID- 21115767 type 1 diabetes leads to hyperglycemia, which is linked to a number of acute (e.g., diabetic ketoacidosis) and chronic (e.g., diabetic nephropathy and cardiovascular disease) complications (1).
PubMedID- 20668157 Close monitoring and frequent insulin dose adjustments in this setting are essential to achieve optimal glycemic control and avoid hypo- or hyperglycemia in patients with type 1 diabetes.
PubMedID- 22187370 type 1 diabetes presents clinically with overt hyperglycemia resulting from progressive immune-mediated destruction of pancreatic beta-cells and associated metabolic dysfunction.
PubMedID- 24741073 Our results show that animals with untreated hyperglycemia in type 1 diabetes mellitus (t1dm) respond to acute insulin challenge much better than the diabetic animals treated with insulin, suggesting that hyperglycemia itself does not cause insulin resistance but treatment of t1dm with insulin does (liu et al.
PubMedID- 20103553 Vinegar decreases postprandial hyperglycemia in patients with type 1 diabetes.
PubMedID- 24592256 This may happen due to one far-off parameter (hyperglycemia in type 1 diabetes (t1d) or high dose glucocorticoid treatment) but is often caused by a sum of risk factors, like the combination: obesity, inactivity, high glycemic index diet, and vitd deficiency.
PubMedID- 20587056 hyperglycemia associated with type 1 diabetes can activate nf-κb [45], which is known to induce b1r [9,10].
PubMedID- 23264286 At the onset, kpd often appears as type 1 diabetes with acute hyperglycemia and ketosis or ketoacidosis and the obvious need for insulin therapy but the signs of autoimmunity against islet β-cells are absent.
PubMedID- 25688339 Background: many studies suggested mesenchymal stem cells (mscs) transplantation as a new approach to control hyperglycemia in type 1 diabetes mellitus through differentiation mechanism.
PubMedID- 24297186 High glucose production contributes to fed and fasted hyperglycemia in type 1 diabetes (t1d) and type 2 diabetes (t2d).
PubMedID- 24843412 Clinically, the pulmonary system appears to be protected from the toxic actions of hyperglycemia because patients with type 1 diabetes are relatively free from vascular pathologies of the pulmonary system.
PubMedID- 22973412 Chronic hyperglycemia exposes patients with type 1 diabetes to an increased risk for death if left untreated.
PubMedID- 22187469 As retinopathy is estimated to take at least 5 years to develop after the onset of hyperglycemia, patients with type 1 diabetes should have an initial dilated and comprehensive eye examination within 5 years after the onset of diabetes.
PubMedID- 20042772 As retinopathy is estimated to take at least 5 years to develop after the onset of hyperglycemia (298), patients with type 1 diabetes should have an initial dilated and comprehensive eye examination within 5 years after the onset of diabetes.
PubMedID- 24721668 Therefore, the longer duration of disease with concomitant longer period of potential hyperglycemia in type 1 diabetes mellitus may be associated with fracture risk independently of bmd.
PubMedID- 22187476 Unexplained hyperglycemia in a patient with type 1 diabetes may suggest impending dka, and monitoring of ketones should therefore be performed.

Page: 1