Disease | hepatitis |
Phenotype | C0020538|hypertension |
Sentences | 9 |
PubMedID- 24694495 | Autoimmune hepatitis in a patient with pulmonary arterial hypertension treated with endothelin receptor antagonists. |
PubMedID- 23635447 | Methodology: patients with portal hypertension due to hepatitis b cirrhosis were analyzed. |
PubMedID- 24651857 | The data for the following variables were reviewed for risk of fanconi syndrome: age, sex, race, history of diabetes or hypertension, co-infection with hepatitis b or hepatitis c, time since hiv diagnosis, time on any antiretroviral therapy, cd4 count, pre-tdf crcl, weight, specific laboratory assessments other than creatinine (alt, ast, albumin, bilirubin, white blood cells, platelets), use of any protease inhibitor (prior, current, and/or ever), use of lopinavir/ritonavir specifically (prior, current, and/or ever), use of atazanavir specifically (prior, current, and/or ever), and duration of tdf receipt. |
PubMedID- 22911697 | [2]–[3] in addition to traditional ckd risk factors such as diabetes and hypertension, co-infection with hepatitis c virus (hcv) has been suggested as a possible risk factor for ckd in hiv-positive individuals. |
PubMedID- 26317657 | Known risk factors for ckd among hiv-positive patients are black race [10], older age, cd4 count <200 cells/mm3, hiv rna levels >4,000 copies/ml, family history of renal disease, clinical progression to aids, diabetes mellitus, hypertension, and co-infection with hepatitis b (hbv) or hepatitis c (hcv) [4]. |
PubMedID- 26205377 | The incidence of portal hypertension (pht) with post-hepatitis cirrhosis is higher in china than elsewhere worldwide. |
PubMedID- 25375659 | Risk of echocardiographic pulmonary hypertension in individuals with human immunodeficiency virus-hepatitis c virus coinfection. |
PubMedID- 22121492 | A platelet count <160,000 × 10(9)/l is 80% sensitive in detecting portal hypertension from cirrhosis in patients with chronic hepatitis c [5]. |
PubMedID- 25673803 | Data was collected on demographic and clinical variables including age, gender, race, alcohol use, tobacco use, etiology of cirrhosis/portal hypertension, infection with hepatitis b or hepatitis c, use of proton pump inhibitors (ppis), use of octreotide, use of antibiotics, and etiology of gastric varices. |
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