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PedAM

Pediatric Disease Annotations & Medicines




Disease glucose intolerance
Phenotype C0028754|adiposity
Sentences 3
PubMedID- 23229739 As might be expected, glucose metabolism followed adiposity, with glucose intolerance in hf fed mice and cycled mice during a hf phase only.
PubMedID- 23667684 In addition, adiopr1-null mice have increased adiposity with augmented glucose intolerance, whereas adipor2-null mice are lean and resistant to diet-induced glucose intolerance [50].
PubMedID- 25960780 The decrease or absence of endogenous melatonin secretion in rats may alter energy metabolism, resulting in increased body weight and visceral adiposity associated with glucose intolerance, insulin resistance, diabetes, dyslipidemia, and cardiovascular disease; melatonin administration in those animals is capable of reverting all the effects mentioned above [7,21-25].

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