Disease | glucose intolerance |
Phenotype | C0011847|diabetes |
Sentences | 38 |
PubMedID- 24719845 | Risk factors for diabetes include previous history of impaired glucose intolerance; high-risk ethnic groups (i.e., asian, african, and hispanic); positive family history or gestational diabetes; age greater than or equal to 45 with either a body mass index (bmi) greater or equal to 25 or presence of hypertension; and established cardiovascular risk factors or disease. |
PubMedID- 23998129 | Overall, the preexisting maternal diabetes leads to glucose intolerance, insulin resistance, and impaired insulin sensitivity and beta -cell function in the offspring at different postnatal periods. |
PubMedID- 21750276 | Women with gdm who develop diabetes face longer periods of glucose intolerance than other glucose-intolerant adults (12), and earlier diagnosis and treatment might be advantageous. |
PubMedID- 24053606 | Since diabetes is a disease of glucose intolerance, most studies have focused on elucidating the role of hyperglycaemia on cv complications. |
PubMedID- 21357466 | In summary, increased dosage of δ40p53 in mice promotes hypoinsulinemia and glucose intolerance, ultimately leading to overt diabetes with age and early death. |
PubMedID- 21257329 | Exposure in utero to maternal diabetes leads to glucose intolerance and high blood pressure with no major effects on lipid metabolism. |
PubMedID- 22974102 | Conclusions: alcohol abstinence might influence diabetes-related factors of alcohol-dependent patients with glucose intolerance. |
PubMedID- 24069200 | Since gestational diabetes is a form of glucose intolerance, it is critical that women with gdm acquire knowledge about foods that contain carbohydrate to facilitate food choices that will not result in hyperglycemia. |
PubMedID- 23298663 | Aims: to assess and compare costs associated with diabetes and lesser degrees of glucose intolerance in australia. |
PubMedID- 24518493 | However, in contrast to type 2 diabetes, onset and progression of glucose intolerance in pancreatic cancer–induced diabetes occur paradoxically in the face of ongoing, often profound, weight loss. |
PubMedID- 22391950 | Several randomised controlled trials have demonstrated that moderate changes in lifestyle including both dietary behaviour and physical activity clearly decrease the risk of progression to diabetes in humans with glucose intolerance. |
PubMedID- 26324089 | In spite of glucose intolerance patients with gck-diabetes showed normal incretin effect, whereas patients with hnf1a-diabetes showed an impaired incretin effect. |
PubMedID- 23319937 | Thus, l-pgds is an important mediator of muscle and adipose glucose transport which is modulated by glycemic conditions and plays a significant role in the glucose intolerance associated with type 2 diabetes [69]. |
PubMedID- 23634949 | Recognizing and treating diabetes or any degree of glucose intolerance in pregnancy results in lowering maternal and fetal complications. |
PubMedID- 23811337 | The kkay mice represent a model for obesity and type 2 diabetes, with spontaneous development of hyperglycemia, hyperinsulinemia, glucose intolerance, and obesity [22,23]. |
PubMedID- 21818460 | Combination of weight loss and exercise to reduce the incidence of type 2 diabetes in patients with glucose intolerance should not be dismissed [5,6]. |
PubMedID- 22936178 | Downregulation of glut4 expression in skm is a major mechanism, which contributes to glucose intolerance associated with type 2 diabetes (31). |
PubMedID- 20862330 | Dysregulated branched-chain metabolism may make an independent contribution to development of insulin resistance and glucose intolerance, ultimately leading to type 2 diabetes [25]. |
PubMedID- 23573479 | Male kk/ta mice spontaneously exhibit type 2 diabetes associated with hyperglycemia, glucose intolerance, hyperinsulinemia, mild obesity and microalbuminuria, conditions which are more severe than those found in the female (2-4). |
PubMedID- 20879973 | We aimed to systematically identify and evaluate those studies that have investigated the impact of lifestyle interventions on the prevention of the development of incident type 2 diabetes in those with glucose intolerance. |
PubMedID- 25977929 | Prepregnancy obesity (30.0+), parity, maternal height, excess pregnancy weight gain, and glucose intolerance without gestational diabetes were significantly associated with a higher infant weight/length ratio in this model. |
PubMedID- 24186867 | Although population studies and experimental models indicate that maternal obesity and diabetes increase risk of obesity and glucose intolerance in offspring, the mechanisms underlying such associations remain unclear. |
PubMedID- 21243857 | The rate of occurrence of variable glucose intolerance was 28% with frank diabetes mellitus diagnosed in 9% of the cases of hyperthyroidism. |
PubMedID- 24899904 | Left ventricular changes are frequently reported in diabetes in existence of glucose intolerance [42]. |
PubMedID- 25898207 | Insulin resistance and islet β-cell dysfunction are important in the pathogenesis of glucose intolerance leading to type 2 diabetes [37]. |
PubMedID- 23878520 | [5] assessed both lifestyle intervention and metformin against placebo intervention in the prevention of type 2 diabetes in individuals with impaired glucose intolerance. |
PubMedID- 26347815 | In addition to being obese, the ob/ob mice are reported to exhibit hyperphagia, a transient diabetes-like syndrome of hyperglycemia, glucose intolerance, elevated plasma insulin, subfertility, impaired wound healing, and increased hormone production from pituitary and adrenal glands, and apparently they are hypometabolic and hypothermic [12]. |
PubMedID- 21194350 | We also observed that reduced synthesis of these selenoproteins caused by overexpression of an i(6)a(-) mutant selenocysteine trna promotes glucose intolerance and leads to a diabetes-like phenotype. |
PubMedID- 23570346 | In previous studies we showed that in japanese-brazilians, low plasma level of total adiponectin is an independent predictor of glucose intolerance and subjects with type 2 diabetes compared with normal glucose tolerant subjects have reduced levels of total adiponectin [14,20]. |
PubMedID- 20304061 | Results from small underpowered trials and post-hoc analyses of data from larger trials designed for bone-specific outcomes show no effect of vitamin d supplementation on glycemia in healthy adults but vitamin d may retard the progression to diabetes in adults with glucose intolerance. |
PubMedID- 23822149 | High admission glucose in patients with diabetes is mainly due to glucose intolerance in the setting of diabetes. |
PubMedID- 21605417 | Both models, b6.cg-m+/+ lepob/j or ob/ob and the b6.v-lepdb/j or db/db), exhibit obesity, hyperphagia, a diabetes-like syndrome of hyperglycemia, glucose intolerance, elevated plasma insulin, and subfertility [37,38]. |
PubMedID- 23928458 | For instance, short sleep duration (<7 hours of sleep per night) and poor sleep quality are associated with cardiovascular morbidity and metabolic disorders such as glucose intolerance, which may lead to obesity, diabetes, heart disease, and hypertension (1). |
PubMedID- 21926287 | Objective the increased risk of type 2 diabetes in women with glucose intolerance in pregnancy is mediated by deterioration of their beta-cell function, which occurs as early as the first year postpartum. |
PubMedID- 23983688 | Gestational diabetes is a diagnosis of any glucose intolerance detected in pregnancy. |
PubMedID- 21494686 | In rodents, models of gestational diabetes produce offspring with glucose intolerance, impaired insulin secretion, insulin resistance and increased adiposity [19], [20], [21]. |
PubMedID- 19788432 | In this review, we discuss current recommendations for managing women with gdm in the postnatal period, with particular attention to postpartum diabetes screening, prevention of future glucose intolerance and family planning. |
PubMedID- 24836607 | In subjects without hiv and without diabetes, the presence of glucose intolerance (ifg or impaired glucose tolerance) is associated with a significant increase in c-imt [37, 38]. |
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