PedAM
Pediatric Disease Annotations & Medicines
| Disease | glomerulonephritis |
| Phenotype | C0027697|nephritis |
| Sentences | 10 |
| PubMedID- 26558120 | In patients with lupus nephritis and biopsy findings of necrotizing and crescentic glomerulonephritis, without significant immune complex deposition, anca testing should be performed. |
| PubMedID- 26393521 | Experimental nephrotoxic nephritis is a model of rapidly progressive glomerulonephritis characterized by: proteinuria; infiltration with macrophages and t-cells; crescent formation in the glomeruli; and th1 antibody (igg2a) and cytokine (il12, and infγ) responses [13]. |
| PubMedID- 21221075 | The c57bl/6 (b6) nephrotoxic nephritis model of crescentic glomerulonephritis (41), the heterozygous fvb/n tg26 hiv-1 (tg26hiv/nl) transgenic mouse model of cg (42), and the b6 kd/kd model of cg (43), have been characterized and described in detail. |
| PubMedID- 23738589 | A kidney biopsy showed acute or chronic tubule-interstitial nephritis with mesangioproliferative glomerulonephritis with no immune deposit on immunofluorescence consistent with lead nephropathy. |
| PubMedID- 21516850 | Aim: to estimate serum and urine il-6 levels and to study its role in diagnosis of nephritis activity in patients with cryoglobulinemic glomerulonephritis (cgn) associated with chronic hepatitis c (chc). |
| PubMedID- 23024773 | The expression values in corresponding controls were arbitrarily set to 1. versican was upregulated 3.5 fold (p = 0.02) in mice with accelerated nephrotoxic serum nephritis, a model of proliferative glomerulonephritis, after 14 days (figure 5). |
| PubMedID- 24574541 | Background: the atp-sensitive p2x7 receptor (p2x7r) has been shown to contribute to renal injury in nephrotoxic nephritis, a rodent model of acute glomerulonephritis, and in unilateral ureteric obstruction (uuo), a rodent model of chronic interstitial inflammation and fibrosis. |
| PubMedID- 23481367 | Lupus-like nephritis, a form of immune complex glomerulonephritis with histologic features identical to lupus nephritis in the absence of clinical or serologic markers of lupus, is well recognized as a cause of end-stage renal disease in hiv-infected patients. |
| PubMedID- 22864120 | Objective: anti-myeloperoxidase anti-neutrophil cytoplasmic antibody (mpo-anca)- related nephritis constitutes 60% of rapidly progressive glomerulonephritis (rpgn) in japan. |
| PubMedID- 23320601 | The indications for plasma exchange was as follows: lupus nephritis with crescentic glomerulonephritis, fibrinoid necrosis and thrombotic microangiopathy in renal pathological changes, severe extra-renal involvement, such as central nervous system injury, hematological abnormalities, cardiovascular diseases, catastrophic antiphospholipid antibody syndrome and so on [14]. |
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