PedAM
Pediatric Disease Annotations & Medicines
| Disease | central pontine myelinolysis |
| Phenotype | C0206083|central pontine myelinolysis |
| Sentences | 16 |
| PubMedID- 23346434 | central pontine myelinolysis (cpm) was first recognized in 1959 by adams et al. |
| PubMedID- 25635862 | No serious adverse events such as central pontine myelinolysis, myocardial infarction, cerebrovascular accident and acute kidney failure were reported in these trials. |
| PubMedID- 26557081 | central pontine myelinolysis (cpm) was originally considered to be the result of excessively rapid correction of slowly progressive hyponatremia in patients with chronic medical conditions, such as chronic alcoholism, malnutrition, and malignancy [1, 2]. |
| PubMedID- 23569555 | central pontine myelinolysis (cpm) is characterized by symmetrical loss of myelin in the basis pontis, with relative preservation of axons and neuronal cell bodies [4]. |
| PubMedID- 21998605 | Patients with anorexia nervosa may be complicated with central pontine myelinolysis in the absence of obvious neurologic deficits by rapid correction8). |
| PubMedID- 26419228 | The central pontine myelinolysis lesions in dogs are virtually identical to those of human cases and the clinical course and manifestations are identical [15]. |
| PubMedID- 23342326 | After a long-term follow up post central pontine myelinolysis after liver transplantation and through a transcranial magnetic stimulation study, we reported that central motor conduction time in the corticospinal tract was delayed. |
| PubMedID- 23546347 | Both for central pontine myelinolysis and wernicke's encephalopathy, mri plays a major role by showing in most cases a suggestive aspect, even if normal imaging does not rule out wernicke's encephalopathy. |
| PubMedID- 21607027 | central pontine myelinolysis (cpm) was first described in 1959 by adams [2] in patients with a history of alcohol abuse. |
| PubMedID- 26044457 | central pontine myelinolysis (cpm) is a neurological disorder caused by damage to the myelin sheath of nerve cells in the brainstem. |
| PubMedID- 22347796 | central pontine myelinolysis (cpm) is a rare neurologic disorder of demyelination that was first reported by adams et al over 50 years ago in a malnourished 38-year-old man with alcohol dependence.1 in addition to malnourishment and alcohol dependence, the adams et al case-report patient was also described as having hyponatremia. |
| PubMedID- 22029026 | [37] the risk of central pontine myelinolysis is lesser in patients with hyperacute hyponatremia that develops rapidly over few hours due to marked increase in water intake, for example, marathon runners, methamphetamine users, etc. |
| PubMedID- 22470870 | There were no cases of central pontine myelinolysis in our patient group. |
| PubMedID- 24741261 | central pontine myelinolysis (cpm) is an uncommon condition with demyelination predominantly in the central portion of the basis pontis. |
| PubMedID- 23326275 | Although adams and his colleagues first described the central pontine myelinolysis (cpm) concept in 1959, the mechanism of ods is not clear until now [1]. |
| PubMedID- 24716023 | central pontine myelinolysis (cpm) is a demyelinating disorder characterized by the loss of myelin in the center of the basis pontis, usually caused by rapid correction of chronic hyponatremia. |
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