PedAM
Pediatric Disease Annotations & Medicines
| Disease | cardiac arrest |
| Phenotype | C0010068|coronary artery disease |
| Sentences | 9 |
| PubMedID- 21658281 | Gwas for discovery and replication of genetic loci associated with sudden cardiac arrest in patients with coronary artery disease. |
| PubMedID- 22359526 | Mutation of this gene is associated with marfan syndrome, loeys-dietz syndrome, familial thoracic aortic aneurysms and dissections and sudden cardiac arrests in patients with coronary artery disease13-15). |
| PubMedID- 25895673 | Since st-elevation is rarely seen and chest pain can often not be assessed in ohca patients [23], guidelines prescribe that cag/pci should be considered in all cardiac arrest patients with suspected coronary artery disease [35]. |
| PubMedID- 23033932 | Cardiac adverse events included 2 episodes of grade 3 systolic dysfunction possibly related to imatinib, and 2 deaths from myocardial infarction and cardiac arrest attributed to pre-existing coronary artery disease. |
| PubMedID- 23015951 | In the london marathon,42 11 of 14 cardiac arrests resulted from coronary artery disease; the other 3 were from unexplained lv hypertrophy or hypertrophic cardiomyopathy. |
| PubMedID- 22360816 | Electrocardiographic predictors of out-of-hospital sudden cardiac arrest in patients with coronary artery disease. |
| PubMedID- 23049969 | The marker snp rs8028182 was found to be associated with sudden cardiac arrest in patients with coronary artery disease [63]; sult1a1, an experimentally validated target of mir-631 [47], has been associated with the risk factor of coronary artery disease [64]. |
| PubMedID- 26064834 | The multicenter unsustained tachycardia trial was initiated in 1989 to test the hypothesis that antiarrhythmic therapy guided by electrophysiologic testing can reduce the risks of sudden death and cardiac arrest among patients with coronary artery disease, lv dysfunction, and spontaneous nsvt. |
| PubMedID- 23938286 | Atherosclerosis susceptibility loci derived from the resistant strains of crosses have been previously detected in at least 3 independent crosses, including a b6×fvb/n ldlr−/− intercross, b6×fvb/n apoe−/− intercross, and a b×h apoe−/− intercross.10,12–13 the chr2 qtl replicated ath28, originally identified in a dba/2.apoe−/−×akr.apoe−/− intercross9 and recently mapped in a b6.apoe−/−×129.apoe−/− intercross.23 the ci of the chr2 qtl (90 to 104 cm) is corresponding to human chr20q13, a region associated with sudden cardiac arrest in patients with coronary artery disease and inflammatory and autoimmunal diseases.31,40–41 the 2 strongest candidate genes for this qtl are lama5 and cdh4. |
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