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PedAM

Pediatric Disease Annotations & Medicines




Disease c syndrome
Phenotype C0022116|ischemia
Sentences 7
PubMedID- 19907345 Microvascular abnormalities caused by endothelial dysfunction seem to be responsible for the myocardial ischemia in patients with cardiac syndrome x (csx).
PubMedID- 24023575 In particular for some silent myocardial ischemia, adopting the method of microcosmic syndrome differentiation can make the treatment more targeted.
PubMedID- 21519976 However, this does not exclude subendocardial ischaemia in this group of patients.32,33 further studies using pet may reveal possible subendocardial ischemia in patients with cardiac syndrome x. hence quantification of regional myocardial blood flow may be clinically useful not only for assessing the extent and severity of coronary vascular disease, but also to detect and measure impairments in microcirculatory function in non-coronary cardiac disease.
PubMedID- 24399243 Background: although increased coronary microvascular resistance (cmr), resulting in coronary microvascular dysfunction, is speculated to be responsible for myocardial ischemia in patients with cardiac syndrome x (csx), it has never been directly demonstrated, and the correlation between cmr and severity of myocardial ischemia has not been elucidated in this setting.
PubMedID- 23129402 We report two cases of acute limb ischemia with threatened myonephropathic metabolic syndrome (mnms) in which continuous hemodiafiltration (chdf) was started before revascularization with selective drainage from the clamped femoral vein of ischemic limb and return of processed blood into the contralateral femoral vein.
PubMedID- 24722851 Aims: the aim of present study was to assess whether vitamin d, with proven beneficial effects on the cardiovascular system, has any effect on angina and exercise-induced ischemia in patients with cardiac syndrome x and low serum vitamin d.
PubMedID- 25511593 Conclusion: in patients of acute lower limb ischemia with myonephropathic metabolic syndrome, early targeted continuous renal replacement therapy may decrease the serum concentrations of myoglobin and ck, improve urine volume, maintain homeostasis, prevent renal function deterioration and improve the prognosis of patients.

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