Disease | c syndrome |
Phenotype | C0022116|ischemia |
Sentences | 7 |
PubMedID- 19907345 | Microvascular abnormalities caused by endothelial dysfunction seem to be responsible for the myocardial ischemia in patients with cardiac syndrome x (csx). |
PubMedID- 24023575 | In particular for some silent myocardial ischemia, adopting the method of microcosmic syndrome differentiation can make the treatment more targeted. |
PubMedID- 21519976 | However, this does not exclude subendocardial ischaemia in this group of patients.32,33 further studies using pet may reveal possible subendocardial ischemia in patients with cardiac syndrome x. hence quantification of regional myocardial blood flow may be clinically useful not only for assessing the extent and severity of coronary vascular disease, but also to detect and measure impairments in microcirculatory function in non-coronary cardiac disease. |
PubMedID- 24399243 | Background: although increased coronary microvascular resistance (cmr), resulting in coronary microvascular dysfunction, is speculated to be responsible for myocardial ischemia in patients with cardiac syndrome x (csx), it has never been directly demonstrated, and the correlation between cmr and severity of myocardial ischemia has not been elucidated in this setting. |
PubMedID- 23129402 | We report two cases of acute limb ischemia with threatened myonephropathic metabolic syndrome (mnms) in which continuous hemodiafiltration (chdf) was started before revascularization with selective drainage from the clamped femoral vein of ischemic limb and return of processed blood into the contralateral femoral vein. |
PubMedID- 24722851 | Aims: the aim of present study was to assess whether vitamin d, with proven beneficial effects on the cardiovascular system, has any effect on angina and exercise-induced ischemia in patients with cardiac syndrome x and low serum vitamin d. |
PubMedID- 25511593 | Conclusion: in patients of acute lower limb ischemia with myonephropathic metabolic syndrome, early targeted continuous renal replacement therapy may decrease the serum concentrations of myoglobin and ck, improve urine volume, maintain homeostasis, prevent renal function deterioration and improve the prognosis of patients. |
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