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PedAM

Pediatric Disease Annotations & Medicines




Disease asthma
Phenotype C0032285|lung inflammation
Sentences 7
PubMedID- 23935767 This level is used as a marker of lung inflammation in patients with asthma, allergic rhinitis, and bronchiectasis to monitor therapy, ie the higher the feno level, the higher the inflammation [13].
PubMedID- 20974000 This short-term model can provide the basis for studying the trophic impact of lodged hmscs on development of lung inflammation associated with acute asthma challenge and the potential benefit of hmscs in circumventing acute asthmatic inflammatory disease.
PubMedID- 22966430 To monitor lung inflammation in patients with bronchial asthma and patients infected with viruses, noninvasive methods have been developed that involve the measurement of exhaled markers, including exhaled nitric oxide (no) [67–71], carbon monoxide (co) [72], volatile gases (e.g., ethane and pentane) [73, 74], and endogenous substances (e.g., inflammatory mediators, cytokines, and oxidants).
PubMedID- 24358127 Allergic airway and lung inflammation, a hallmark of bronchial asthma, represents a complex pathological condition that is characterized by bronchial hyper-responsiveness, airway eosinophilia, and macrophage activation.
PubMedID- 26482437 In conclusion, this study shows that β-catenin expression regulates the differentiation of inkt cells that augment lung inflammation associated with asthma and provides insights that may be of use in designing regimens to limit at least one type of lung inflammation.
PubMedID- 24492762 Evidence of human toxicity ofnanoparticles, for example, oxidative damage, lung inflammation, asthma, possibly lungcancer, atherosclerosis, and worsening of heart disease, came from epidemiological studiesof unintentionally or naturally produced ultrafine particles generated from trafficpollution and combustion processes such as diesel exhaust and welding fumes4,5,6,7,8,9).epidemiological studies have shown positive correlation between the particulate matter inair pollution and increased morbidity and mortality in adults and children4, 5).epidemiological studies also show links between respiratory illnesses and the number ofambient ultrafine particles4, 5).
PubMedID- 23165884 Respiratory infections by human rhinovirus account for most asthma exacerbations, with virus-induced lung inflammation and lung damage directly related to loss of lung function.

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