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PedAM

Pediatric Disease Annotations & Medicines




Disease arteriosclerosis
Phenotype C0948265|metabolic syndrome
Sentences 44
PubMedID- 21321616 [detection of the parameters for early atherosclerosis in patients with metabolic syndrome and periodontitis].
PubMedID- 25247758 metabolic syndrome is correlated with carotid atherosclerosis in patients with lupus nephritis.
PubMedID- 24490696 Tempol attenuates atherosclerosis associated with metabolic syndrome via decreased vascular inflammation and nadph-2 oxidase expression.
PubMedID- 24072083 Carotid intima-media thickness (imt) is a potential indicator of subclinical atherosclerosis in patients with metabolic syndrome (ms).
PubMedID- 22383927 El plasma concentrations are increased in metabolic syndrome and associated with coronary atherosclerosis [3].
PubMedID- 26089893 Using the liver insulin receptor knockout (lirko) mouse as a model of pure hepatic insulin resistance it has been demonstrated that hepatic insulin resistance alone can produce both the dyslipidemia and increasing risk of atherosclerosis associated with the metabolic syndrome [51].
PubMedID- 23409033 Implication of progranulin and c1q/tnf-related protein-3 (ctrp3) on inflammation and atherosclerosis in subjects with or without metabolic syndrome.
PubMedID- 20727144 Subclinical carotid atherosclerosis is associated with metabolic syndrome [38].
PubMedID- 20470376 Association between serum levels of incretins, glucagon-like peptide-1 (glp-1) and gastric inhibitory polypeptide (gip), and metabolic syndrome components in patients with atherosclerosis-prone conditions.
PubMedID- 21738343 Differential relationship between metabolic syndrome score and severity of coronary atherosclerosis as assessed by angiography in a non-diabetic and diabetic korean population.
PubMedID- 21827549 Background: carotid intima-media thickness (cimt) is a potential indicator of subclinical atherosclerosis in patients with metabolic syndrome (mets).
PubMedID- 24481091 [relation between mean platelet volume and subclinical atherosclerosis in patients with metabolic syndrome].
PubMedID- 24843601 Adipocytokines play an important role in the pathogenesis of atherosclerosis through inflammation associated with obesity and metabolic syndrome.
PubMedID- 23448759 Significance of the presence of metabolic syndrome in patients with asymptomatic arteriosclerosis affecting the aorta and the cerebral, extra-cranial carotid and coronary arteries.
PubMedID- 25218813 Circulating osteoprotegerin is increased in the metabolic syndrome and associates with subclinical atherosclerosis and coronary arterial calcification.
PubMedID- 21816063 Furthermore, our findings suggest that the observed association between uric acid and carotid atherosclerosis may be attributed to metabolic syndrome-dependent and -independent mechanisms.
PubMedID- 22493605 Background: the renin-angiotensin-aldosterone system plays an important role in the development and progression of hypertension and accelerated atherosclerosis (atheroscleropathy) associated with the cardiorenal metabolic syndrome and type 2 diabetes mellitus.
PubMedID- 23798936 A recent population-based study reported that the association of a metabolic syndrome with subclinical atherosclerosis was independent of its association with the biomarkers of endothelial damage and oxidative stress.
PubMedID- 23398954 Associations of cxcl16/cxcr6 with carotid atherosclerosis in patients with metabolic syndrome.
PubMedID- 21497934 Aim: the goal of this study was to investigate the association between omentin-1 and carotid atherosclerosis in patients with metabolic syndrome (mets).
PubMedID- 26381729 Recent studies suggested that uoc could serve as a novel biomarker for carotid atherosclerosis in patients with hypertension or metabolic syndrome [7, 8].
PubMedID- 25633268 Aims: matrix metalloproteinases (mmps) and their tissue inhibitors (timps) are dysregulated in metabolic syndrome (mets) and associated with atherosclerosis and cardiovascular disease (cvd).
PubMedID- 25233795 Association of metabolic syndrome with coronary atherosclerosis in non-diabetic postmenopausal women.
PubMedID- 23773268 Dysregulation of adipocytokines caused by visceral fat accumulation is regarded as one of the major pathophysiological mechanisms of atherosclerosis associated with the metabolic syndrome.
PubMedID- 20398632 Background: oxidative stress induced by free fatty acids plays a critical role in the pathogenesis of endothelial dysfunction and atherosclerosis in patients with metabolic syndrome.
PubMedID- 26141940 The aim of this study is to evaluate the impact of fabp4 polymorphisms on fabp4 plasma levels and subclinical arteriosclerosis in patients with obesity, metabolic syndrome (ms) or type 2 diabetes (t2dm).
PubMedID- 25206497 It is also a specific biomarker for atherosclerosis-prone patients with metabolic syndrome[33].
PubMedID- 22919126 This suggests that vascular atherosclerosis resulting from metabolic syndrome may be part of the pathophysiology of ed in men with hypogonadism.
PubMedID- 25242580 Therefore, the intervention with anti-inflammatory agents may effectively delay the formation and progression of atherosclerosis in patients with metabolic syndrome.
PubMedID- 21772967 Based on this data and the sparcl (stroke prevention by aggressive reduction in cholesterol levels) trial [75], the recommendation is for aggressive lipid lowering with statin in patients with atherosclerotic ischemic stroke.another wasid substudy [76] has demonstrated that metabolic syndrome is associated with intracranial atherosclerosis and confers a higher risk of major vascular events in these patients.
PubMedID- 20689417 Recent findings: individuals infected with hiv frequently demonstrate a metabolic syndrome associated with atherosclerosis and cardiovascular disease.
PubMedID- 19762024 Objective: metabolic syndrome (ms) is associated with subclinical atherosclerosis, but the relative role of obstructive sleep apnoea (osa) is largely unknown.
PubMedID- 23776788 [16] il-18 has been shown to be highly expressed in atherosclerotic plaques, mainly in plaque macrophages, and in particular in unstable plaques,[17] and the circulating il-18 level is a useful biomarker for atherosclerosis prone patients with metabolic syndrome.
PubMedID- 24470352 Also observed defects in akt2 signaling which may contribute to accelerated atherosclerosis in patients with metabolic syndrome (18).
PubMedID- 21190590 In our previous study of a specific atherosclerosis-prone population with obesity and metabolic syndrome (mets), we did not find any significant role for at-ldl [19].
PubMedID- 25589838 metabolic syndrome criteria as predictors of subclinical atherosclerosis based on the coronary calcium score.
PubMedID- 20039893 The metabolic syndrome is associated with subclinical atherosclerosis independent of insulin resistance: the guangzhou biobank cohort study-cvd.
PubMedID- 21251304 Circulating interleukin-18: a specific biomarker for atherosclerosis-prone patients with metabolic syndrome.
PubMedID- 22813569 Association of metabolic syndrome with carotid atherosclerosis in the young north indian population.
PubMedID- 23704519 Human epicardial fat in type 2 diabetic and metabolic syndrome patients with coronary atherosclerosis (cad) expressed amounts of ucp1 similar to those expressed in control subjects without diabetes, metabolic syndrome, and cad (20).
PubMedID- 22738646 The metabolic syndrome and progression of carotid atherosclerosis over 13 years.
PubMedID- 22291799 In our study, the level of inflammatory markers crp and tnf-α, agents of the coagulation system activation and fibrinolysis (d-dimers, fibrinogen) participating in the inflammatory process of atherosclerosis were higher in patients with the metabolic syndrome as compared with healthy women.
PubMedID- 23245314 Decreased serum obestatin consequent upon trib3 q84r polymorphism exacerbates carotid atherosclerosis in subjects with metabolic syndrome.
PubMedID- 25546822 Interestingly, therefore, myd88-dependent cooperation between myeloid and endothelial cells was recently proven to be key to the promotion of the vascular inflammation and atherosclerosis associated with metabolic syndrome 64. thus, because myd88 is likely to be a key player in integrating cross-talk among tlr, fcr, and complement receptors, in concert with the reduction in igg and c3a deposition, our finding that es-62 and anti–il-22 down-regulate myd88 expression in kidney cells provides an effective mechanism for protecting against kidney inflammation.

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