Disease | arteriosclerosis |
Phenotype | C0948265|metabolic syndrome |
Sentences | 44 |
PubMedID- 21321616 | [detection of the parameters for early atherosclerosis in patients with metabolic syndrome and periodontitis]. |
PubMedID- 25247758 | metabolic syndrome is correlated with carotid atherosclerosis in patients with lupus nephritis. |
PubMedID- 24490696 | Tempol attenuates atherosclerosis associated with metabolic syndrome via decreased vascular inflammation and nadph-2 oxidase expression. |
PubMedID- 24072083 | Carotid intima-media thickness (imt) is a potential indicator of subclinical atherosclerosis in patients with metabolic syndrome (ms). |
PubMedID- 22383927 | El plasma concentrations are increased in metabolic syndrome and associated with coronary atherosclerosis [3]. |
PubMedID- 26089893 | Using the liver insulin receptor knockout (lirko) mouse as a model of pure hepatic insulin resistance it has been demonstrated that hepatic insulin resistance alone can produce both the dyslipidemia and increasing risk of atherosclerosis associated with the metabolic syndrome [51]. |
PubMedID- 23409033 | Implication of progranulin and c1q/tnf-related protein-3 (ctrp3) on inflammation and atherosclerosis in subjects with or without metabolic syndrome. |
PubMedID- 20727144 | Subclinical carotid atherosclerosis is associated with metabolic syndrome [38]. |
PubMedID- 20470376 | Association between serum levels of incretins, glucagon-like peptide-1 (glp-1) and gastric inhibitory polypeptide (gip), and metabolic syndrome components in patients with atherosclerosis-prone conditions. |
PubMedID- 21738343 | Differential relationship between metabolic syndrome score and severity of coronary atherosclerosis as assessed by angiography in a non-diabetic and diabetic korean population. |
PubMedID- 21827549 | Background: carotid intima-media thickness (cimt) is a potential indicator of subclinical atherosclerosis in patients with metabolic syndrome (mets). |
PubMedID- 24481091 | [relation between mean platelet volume and subclinical atherosclerosis in patients with metabolic syndrome]. |
PubMedID- 24843601 | Adipocytokines play an important role in the pathogenesis of atherosclerosis through inflammation associated with obesity and metabolic syndrome. |
PubMedID- 23448759 | Significance of the presence of metabolic syndrome in patients with asymptomatic arteriosclerosis affecting the aorta and the cerebral, extra-cranial carotid and coronary arteries. |
PubMedID- 25218813 | Circulating osteoprotegerin is increased in the metabolic syndrome and associates with subclinical atherosclerosis and coronary arterial calcification. |
PubMedID- 21816063 | Furthermore, our findings suggest that the observed association between uric acid and carotid atherosclerosis may be attributed to metabolic syndrome-dependent and -independent mechanisms. |
PubMedID- 22493605 | Background: the renin-angiotensin-aldosterone system plays an important role in the development and progression of hypertension and accelerated atherosclerosis (atheroscleropathy) associated with the cardiorenal metabolic syndrome and type 2 diabetes mellitus. |
PubMedID- 23798936 | A recent population-based study reported that the association of a metabolic syndrome with subclinical atherosclerosis was independent of its association with the biomarkers of endothelial damage and oxidative stress. |
PubMedID- 23398954 | Associations of cxcl16/cxcr6 with carotid atherosclerosis in patients with metabolic syndrome. |
PubMedID- 21497934 | Aim: the goal of this study was to investigate the association between omentin-1 and carotid atherosclerosis in patients with metabolic syndrome (mets). |
PubMedID- 26381729 | Recent studies suggested that uoc could serve as a novel biomarker for carotid atherosclerosis in patients with hypertension or metabolic syndrome [7, 8]. |
PubMedID- 25633268 | Aims: matrix metalloproteinases (mmps) and their tissue inhibitors (timps) are dysregulated in metabolic syndrome (mets) and associated with atherosclerosis and cardiovascular disease (cvd). |
PubMedID- 25233795 | Association of metabolic syndrome with coronary atherosclerosis in non-diabetic postmenopausal women. |
PubMedID- 23773268 | Dysregulation of adipocytokines caused by visceral fat accumulation is regarded as one of the major pathophysiological mechanisms of atherosclerosis associated with the metabolic syndrome. |
PubMedID- 20398632 | Background: oxidative stress induced by free fatty acids plays a critical role in the pathogenesis of endothelial dysfunction and atherosclerosis in patients with metabolic syndrome. |
PubMedID- 26141940 | The aim of this study is to evaluate the impact of fabp4 polymorphisms on fabp4 plasma levels and subclinical arteriosclerosis in patients with obesity, metabolic syndrome (ms) or type 2 diabetes (t2dm). |
PubMedID- 25206497 | It is also a specific biomarker for atherosclerosis-prone patients with metabolic syndrome[33]. |
PubMedID- 22919126 | This suggests that vascular atherosclerosis resulting from metabolic syndrome may be part of the pathophysiology of ed in men with hypogonadism. |
PubMedID- 25242580 | Therefore, the intervention with anti-inflammatory agents may effectively delay the formation and progression of atherosclerosis in patients with metabolic syndrome. |
PubMedID- 21772967 | Based on this data and the sparcl (stroke prevention by aggressive reduction in cholesterol levels) trial [75], the recommendation is for aggressive lipid lowering with statin in patients with atherosclerotic ischemic stroke.another wasid substudy [76] has demonstrated that metabolic syndrome is associated with intracranial atherosclerosis and confers a higher risk of major vascular events in these patients. |
PubMedID- 20689417 | Recent findings: individuals infected with hiv frequently demonstrate a metabolic syndrome associated with atherosclerosis and cardiovascular disease. |
PubMedID- 19762024 | Objective: metabolic syndrome (ms) is associated with subclinical atherosclerosis, but the relative role of obstructive sleep apnoea (osa) is largely unknown. |
PubMedID- 23776788 | [16] il-18 has been shown to be highly expressed in atherosclerotic plaques, mainly in plaque macrophages, and in particular in unstable plaques,[17] and the circulating il-18 level is a useful biomarker for atherosclerosis prone patients with metabolic syndrome. |
PubMedID- 24470352 | Also observed defects in akt2 signaling which may contribute to accelerated atherosclerosis in patients with metabolic syndrome (18). |
PubMedID- 21190590 | In our previous study of a specific atherosclerosis-prone population with obesity and metabolic syndrome (mets), we did not find any significant role for at-ldl [19]. |
PubMedID- 25589838 | metabolic syndrome criteria as predictors of subclinical atherosclerosis based on the coronary calcium score. |
PubMedID- 20039893 | The metabolic syndrome is associated with subclinical atherosclerosis independent of insulin resistance: the guangzhou biobank cohort study-cvd. |
PubMedID- 21251304 | Circulating interleukin-18: a specific biomarker for atherosclerosis-prone patients with metabolic syndrome. |
PubMedID- 22813569 | Association of metabolic syndrome with carotid atherosclerosis in the young north indian population. |
PubMedID- 23704519 | Human epicardial fat in type 2 diabetic and metabolic syndrome patients with coronary atherosclerosis (cad) expressed amounts of ucp1 similar to those expressed in control subjects without diabetes, metabolic syndrome, and cad (20). |
PubMedID- 22738646 | The metabolic syndrome and progression of carotid atherosclerosis over 13 years. |
PubMedID- 22291799 | In our study, the level of inflammatory markers crp and tnf-α, agents of the coagulation system activation and fibrinolysis (d-dimers, fibrinogen) participating in the inflammatory process of atherosclerosis were higher in patients with the metabolic syndrome as compared with healthy women. |
PubMedID- 23245314 | Decreased serum obestatin consequent upon trib3 q84r polymorphism exacerbates carotid atherosclerosis in subjects with metabolic syndrome. |
PubMedID- 25546822 | Interestingly, therefore, myd88-dependent cooperation between myeloid and endothelial cells was recently proven to be key to the promotion of the vascular inflammation and atherosclerosis associated with metabolic syndrome 64. thus, because myd88 is likely to be a key player in integrating cross-talk among tlr, fcr, and complement receptors, in concert with the reduction in igg and c3a deposition, our finding that es-62 and anti–il-22 down-regulate myd88 expression in kidney cells provides an effective mechanism for protecting against kidney inflammation. |
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