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PedAM

Pediatric Disease Annotations & Medicines




Disease arteriosclerosis
Phenotype C0011849|diabetes mellitus
Sentences 69
PubMedID- 22022185 It has been shown that atherosclerosis in patients with diabetes mellitus (dm) can lead to acute coronary artery disease, ischemic cerebral disease, and peripheral arterial disease (2).
PubMedID- 22576258 The pathophysiology of atherosclerosis development in patients with diabetes mellitus (dm) is similar to that in nondiabetics.
PubMedID- 23904855 In this study, the total number for atherosclerosis in patients with type 2 diabetes mellitus increased with the progression of renal dysfunction.
PubMedID- 21957962 This may contribute to atherosclerosis in diabetes mellitus.
PubMedID- 25120969 Effect of age and blood pressure on surrogate markers of atherosclerosis in patients with type 2 diabetes mellitus.
PubMedID- 22421230 Objectives: the goal of this study was to characterize the extent and composition of coronary atherosclerosis in patients with diabetes mellitus or the metabolic syndrome (met syn) presenting with acute coronary syndromes (acs).
PubMedID- 25253014 Conclusion: the systolic longitudinal myocardial function of lv become compromised before reduced lv global systolic function in coronary atherosclerosis patients with type 2 diabetes mellitus.
PubMedID- 25875738 However, little is known about the association between glycemic control and objective sleep architecture and its influence on arteriosclerosis in patients with type-2 diabetes mellitus (dm).
PubMedID- 19853860 Objective: to compare aortic intima media thickness (aimt) to carotid intima media thickness (cimt) as a marker of early atherosclerosis in children with type 1 diabetes mellitus and to examine the associations of aimt to known cardiovascular risk factors.
PubMedID- 22394699 Association between apolipoprotein e polymorphism and subclinic atherosclerosis in patients with type 1 diabetes mellitus.
PubMedID- 22368195 Aims: several studies have demonstrated that type 2 diabetes mellitus (t2dm) is associated with accelerated atherosclerosis, which results in an increased risk of coronary vascular events.
PubMedID- 20593206 The objective of this study is to investigate the correlation of urinary albumin excretion rate (uaer) with the incidence of coronary heart disease (chd), pathological characteristics and severity of coronary atherosclerosis in patients with type 2 diabetes mellitus (t2dm), and explore the efficacy of using the urinary albumin excretion rate (uaer) to predict the risk of chd in patients with t2dm.
PubMedID- 26198368 Accelerated atherosclerosis in patients with diabetes mellitus contributes an increased risk of developing cardiovascular diseases including peripheral vascular disease (pvd).
PubMedID- 24988089 This might be one of the causal pathogenic factors initiating accelerated atherosclerosis in patients with type 2 diabetes mellitus.
PubMedID- 26345606 Therefore, serms may also be effective in reducing the progression of arteriosclerosis in patients with diabetes mellitus.
PubMedID- 25975128 [a relationship between cardiac structural and functional parameters, left ventricular contractility, and coronary atherosclerosis in patients with type 2 diabetes mellitus].
PubMedID- 24593955 Altered lipids and lipoprotein metabolism in chronic diabetes mellitus is associated with pathogenesis of atherosclerosis and other cardiovascular diseases.
PubMedID- 25456820 High-intensity statin therapy and regression of coronary atherosclerosis in patients with diabetes mellitus.
PubMedID- 20194881 Effect of rosiglitazone on progression of coronary atherosclerosis in patients with type 2 diabetes mellitus and coronary artery disease: the assessment on the prevention of progression by rosiglitazone on atherosclerosis in diabetes patients with cardiovascular history trial.
PubMedID- 20516379 The phosphodiesterase inhibitor cilostazol induces regression of carotid atherosclerosis in subjects with type 2 diabetes mellitus: principal results of the diabetic atherosclerosis prevention by cilostazol (dapc) study: a randomized trial.
PubMedID- 22120969 Glp-1 may prevent or delay the formation of atherosclerosis in diabetes mellitus by improving the function of enos.
PubMedID- 23574730 Postprandial hyperglycemia and/or hyperlipidemia can contribute to development of atherosclerosis in patients with type 2 diabetes mellitus (t2dm).
PubMedID- 24797048 The present study was designed to investigate the association of polymorphisms xbai (rs693) and ecori (rs1042031) of the apob gene with plasma apob level, lipid levels and the different ultrasound phenotypes of carotid atherosclerosis in patients with diabetes mellitus type 2.
PubMedID- 24768387 Rationale and design of a study to evaluate the effects of sitagliptin on atherosclerosis in patients with diabetes mellitus: prologue study.
PubMedID- 24423050 The gstt1-null genotype and combined gstt1*0/gstm1*0 might be potential determinants of susceptibility to advanced atherosclerosis in patients with type 2 diabetes mellitus [16].
PubMedID- 25708055 In conclusion, cumulative active and passive smoking exposures are significant risk factors for carotid atherosclerosis in patients with type 2 diabetes mellitus.
PubMedID- 22330623 The gstt1-0 genotype and gstt1-0/gstm1-0 haplotype might be a potential determinants of susceptibility to advanced atherosclerosis in patients with type 2 diabetes mellitus.
PubMedID- 24392103 While pwv is affected by changes in instantaneous blood pressure, the cardio-ankle vascular index (cavi) is a blood pressure-independent index of systemic arterial stiffness, and is often used as a marker of early arteriosclerosis associated with hypertension, diabetes mellitus, dyslipidemia and smoking [5].
PubMedID- 23230750 According to the levels of cimt, 64 diabetic patients were classified into two groups: diabetes mellitus without atherosclerosis (a group, n = 37) and diabetes mellitus with atherosclerosis (b group, n = 27).
PubMedID- 20605247 According to the levels of carotid intima-media thickness (cimt), 36 diabetic patients were classified into two groups, the diabetes mellitus without atherosclerosis (dm-as, n=20) and diabetes mellitus with atherosclerosis (dm+as, n=16).
PubMedID- 20004424 We investigated the relationship between hemoglobin concentration and various factors as well as markers of subclinical atherosclerosis in men with type 2 diabetes mellitus.
PubMedID- 20538123 Comparison of rates of progression of coronary atherosclerosis in patients with diabetes mellitus versus those with the metabolic syndrome.
PubMedID- 25509887 Aim: to study the prognostic value of multifocal atherosclerosis (mfa) in patients with diabetes mellitus (dm) at high risk for myocardial ischemia who need coronary angiography (cag).
PubMedID- 23678326 Chronic subclinical inflammation is considered to be important for the initiation and / or progression of atherosclerosis in patients with diabetes mellitus.
PubMedID- 25851542 [33] suggested that ga was a more valuable index than hba1c for predicting the progression of atherosclerosis in patients with type 2 diabetes mellitus.
PubMedID- 21996253 Aims: to analyze the relationship between serum levels of osteocalcin and parameters of atherosclerosis in patients with type 2 diabetes mellitus (t2dm).
PubMedID- 24678948 The aim of this study was to investigate the relationships of subcutaneous and visceral abdominal fat with carotid atherosclerosis in patients with type 2 diabetes mellitus (t2dm).
PubMedID- 22827403 The aim of our study was to evaluate serum amyloid a (saa), an acute phase reactant, and carotid intima-media thickness (cimt) as a valid predictor of atherosclerosis in women with gestational diabetes mellitus (gdm).
PubMedID- 26069232 Little data exist about the relationship of apoc-iii, triglycerides, and atherosclerosis in patients with type 2 diabetes mellitus (t2dm).
PubMedID- 20361178 This pathogenic sequence establishes the molecular basis linking insulin resistance, inflammation and accelerated atherosclerosis in people with type 2 diabetes mellitus and may help account for the missing 30% cvd risk that cannot be explained by circulating cardiovascular risk factors [74, 76] (fig.
PubMedID- 26505665 Method: this case-control study included 596 type 2 diabetes mellitus patients with carotid atherosclerosis and 379 patients without carotid atherosclerosis.
PubMedID- 24739810 Increased levels of adma were found in the development of carotid atherosclerosis in patients with type 2 diabetes mellitus (t2dm) [22], in subjects with early asymptomatic carotid atherosclerosis [23], and especially in patients with chronic kidney disease (ckds) and rheumatoid arthritis [24].
PubMedID- 26251624 The development of atherosclerosis in patients with type 2 diabetes mellitus (t2dm) may be due to hypercoagulability and platelet hyperaggregability,1,2 along with increased levels of platelet activation markers.
PubMedID- 23347332 Conclusions: diabetes mellitus may be associated with subclinical atherosclerosis assessed by measurement of avp and cimt.
PubMedID- 25385870 It is well known that hyperglycemia is a trigger of atherosclerosis in patients with diabetes mellitus.
PubMedID- 21483695 Part of the increased susceptibility to atherosclerosis of patients with type 2 diabetes mellitus and the metabolic syndrome has been attributed to the decreased clearance of trps [5], [31], [32].
PubMedID- 21747057 Background: endothelial dysfunction contributes to the development of atherosclerosis in patients with diabetes mellitus, but the mechanisms of endothelial dysfunction in this setting are incompletely understood.
PubMedID- 25537059 Background: we aimed to investigate the association of toenail onychomycosis with subclinical atherosclerosis in patients with diabetes mellitus.
PubMedID- 20108050 Since postprandial lipoproteins are atherogenic, the delay in clearance of these particles could play an important role in the development of atherosclerosis in patients with diabetes mellitus.
PubMedID- 21779189 Leptin and adiponectin can be more relevant to atherosclerosis in subjects with obesity and diabetes mellitus than conventional atherosclerotic factors such as bp and lipid levels [7, 14, 15].

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