PedAM
Pediatric Disease Annotations & Medicines
| Disease | anoxia |
| Phenotype | C0520679|obstructive sleep apnea |
| Sentences | 14 |
| PubMedID- 26189914 | Urinary uric acid excretion as an indicator of severe hypoxia and mortality in patients with obstructive sleep apnea and chronic obstructive pulmonary disease. |
| PubMedID- 23174804 | Chronic intermittent hypoxia (cih) occurs with obstructive sleep apnea syndrome (osas) and provokes systemic endothelial dysfunction, which is associated with oxidative stress and low nitric oxide (no) bioavailability. |
| PubMedID- 21245593 | Consequences of hypoxia-reoxygenation phenomena in patients with obstructive sleep apnea syndrome. |
| PubMedID- 25369321 | obstructive sleep apnea (osa) associated with chronic intermittent hypoxia (cih) increases the morbidity and mortality of ischemic heart disease in patients. |
| PubMedID- 25333334 | obstructive sleep apnea: role of intermittent hypoxia and inflammation. |
| PubMedID- 25364071 | Conclusions: these results suggest that the intermittent hypoxia associated with obstructive sleep apnea (osa) could induce fertility reduction in male patients with this sleep breathing disorder. |
| PubMedID- 23826169 | Introduction: recurrent hypoxia, which is associated with obstructive sleep apnea syndrome (osas), leads to an increase in the degradation of adenosine triphosphatase into xanthine, which in turn increases uric acid concentrations. |
| PubMedID- 25573199 | Also, obstructive sleep apnea with hypoxia in obese people is associated with insulin resistance, metabolic syndrome, and cardiovascular abnormalities [41,42]. |
| PubMedID- 24713530 | Background and purpose: obstructive sleep apnea, a condition associated with chronic intermittent hypoxia (cih), carries an increased risk of stroke. |
| PubMedID- 23259297 | Objective: to observe the levels of nitrogen oxide (no), vascular endothelial growth factor (vegf) and hypoxia-inducible factor-1alpha (hif-1alpha) in patients with obstructive sleep apnea-hypopnea syndrome (osahs) and to discuss their roles in the pathogenesis of cardiovascular disease in patients with osahs. |
| PubMedID- 26429743 | Background: obstructive sleep apnea (osa) leads to intermittent hypoxia, activation of the sympathetic nervous system, and eventually cardiovascular morbidity. |
| PubMedID- 26102761 | Objective: recurrent apnea and hypoxia, which is associated with obstructive sleep apnea syndrome (osas), leads to an increase in the degradation of adenosine triphosphatase (atp) into xanthine, which in turn increases uric acid (ua) concentrations. |
| PubMedID- 21429415 | Objective: chronic intermittent hypoxia (cih) occurs in patients with obstructive sleep apnea-hypopnea syndrome (osahs) and has adverse effects on multiple physiological functions. |
| PubMedID- 24229461 | Dr. dempsey presented neutral views from the chair's perspective, basically highlighting how intermittent hypoxia exposure (with reference to obstructive sleep apnea and healthy human models of it) affects cardiovascular system. |
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