Disease | anoxia |
Phenotype | C0024115|pulmonary disease |
Sentences | 8 |
PubMedID- 21216882 | Rationale: since the introduction of long-term oxygen treatment (ltot) in chronic obstructive pulmonary disease (copd) with chronic hypoxia, the proportion of women and the age of patients starting ltot have increased markedly. |
PubMedID- 25851169 | Conclusions: the mechanism described here adds to our knowledge of the pathogenesis of severe pulmonary diseases associated with hypoxia, e.g. |
PubMedID- 20345439 | Purpose: to evaluate whether patent foramen ovale (pfo) is a contributing factor to hypoxia in patients with chronic obstructive pulmonary disease (copd). |
PubMedID- 25902085 | Background: in a clinical study, the role of chronic hypoxia in patients with chronic obstructive pulmonary disease or the role of transient hypoxia in patients with obstructive sleep apnea syndrome were examined for the peripapillary retinal nerve fiber layers. |
PubMedID- 23549866 | The most notable toxicity was grade 3 pericardial effusion, which gradually increased 2 years after treatment start until discontinuation of the trial resulting in pericardial tamponade, grade 3 hypoxia associated with chronic obstructive pulmonary disease (copd), which he had before the trial, and latent interstitial pneumonia. |
PubMedID- 21397736 | Air travel may cause significant hypoxia in passengers with chronic obstructive pulmonary disease (copd). |
PubMedID- 22798194 | Recently we observed increased adipose tissue (at) expression of cd40-related signaling proteins but no activation of tumor necrosis factor-alpha or cd68 in patients with chronic sustained hypoxia resulting from chronic obstructive pulmonary disease (copd). |
PubMedID- 24454367 | Both long-term activation of inflammation and chronic pulmonary disease, with dyspnea and hypoxia, have been shown to decrease igf-i levels and to compromise children's growth, and these findings have been demonstrated in children with cystic fibrosis [8, 9]. |
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