PedAM
Pediatric Disease Annotations & Medicines
| Disease | anoxia |
| Phenotype | C0020456|hyperglycemia |
| Sentences | 10 |
| PubMedID- 25193495 | Under hyperglycemia combined with hypoxia up to 12 hr, the expression of rplp0, tfrc, gusb, and actb genes remained unchanged. |
| PubMedID- 22125614 | Revealed that sensory and neurosecretory nociceptor functions are sensitized in diabetic rats and hyperglycemia with hypoxia increased the sensitivity of diabetic c-fibers more than hyperglycemia alone [47] suggesting that not only the correction of diabetes but also the recovery from hypoxia is a valuable target for the treatment of painful diabetic neuropathy. |
| PubMedID- 20182531 | The characteristic microvascular damage caused by hyperglycemia [50] may lead to periods of hypoxia, possibly through a nitric-oxide-mediated mechanism. |
| PubMedID- 22754875 | [31] studies with fetal blood sampling confirm that hyperglycemia has been associated with fetal hypoxia and acidosis. |
| PubMedID- 26221572 | Nerves that were subjected to hyperglycemia (55.5 mmol/l) with anoxia showed more dramatic pathological changes than nerves that underwent anoxia at normoglycemia. |
| PubMedID- 26074963 | Other studies have demonstrated that hyperglycemia worsens the impact of ischemia and hypoxia, leading to worse outcomes [25, 26]. |
| PubMedID- 24278186 | hyperglycemia induces hypoxia in retinal tissues, thus leading to the production of vegf for the neovascularization in diabetic retinopathy [10]. |
| PubMedID- 20127827 | The purpose of this study was to evaluate the hypothesis that in the developing embryo hyperglycemia also leads to hypoxia, both resulting in oxidative damage. |
| PubMedID- 24564828 | hypoxia may interact with hyperglycemia and promote diabetes and its complications. |
| PubMedID- 25050232 | There are multiple changes in diabetic patients that are associated with oxidative stress such as: glycation of some products (diabetes care, 2010 ▶) and hypoxia resulting from hyperglycemia (kaneto et al., 2007 ▶) that can make an imbalance in cellular oxidation and reduction, especially in liver tissue (gallou et al., 2007 ▶). |
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