Home Contact Sitemap

PedAM

Pediatric Disease Annotations & Medicines




Disease proteinuria
Symptom C0268731|glomerular disease
Sentences 14
PubMedID- 23418489 Alterations of podocyte ultrastructure, in particular its foot processes, are the histologic hallmark of glomerular disease associated with severe proteinuria, and are closely linked to disaggregation and altered distribution of actin filaments 2.
PubMedID- 20838616 Podocyte foot process effacement is commonly observed in a large variety of glomerular diseases associated with proteinuria 3.
PubMedID- 23232022 Focal segmental glomerulosclerosis (fsgs) is a form of glomerular disease associated with proteinuria and nephritic syndrome.
PubMedID- 23817053 Podocyte injury is crucial in the pathogenesis of glomerular disease with proteinuria, leading to ckd.
PubMedID- 22190940 Foot process effacement can be observed in a variety of human and experimental glomerular diseases associated with massive proteinuria, including minimal change disease, focal segmental glomerulosclerosis (fsgs), membranous glomerulopathy, iga-nephropathy, diabetic nephropathy, and lupus nephritis .
PubMedID- 25886386 Furthermore, accumulating evidence indicates that er stress contributes to glomerular diseases with proteinuria .
PubMedID- 25866774 Local production of angiotensin ii (ang ii), the main component of the renin-angiotensin system, is upregulated in glomerular disease with proteinuria .
PubMedID- 24635376 Objective: to seek consensus among renal specialists for the evaluation of dogs with proteinuria because of suspected glomerular disease.
PubMedID- 25003154 The most relevant clinicopathological findings were thrombocytopenia, hypoalbuminemia, and normal azotemic proteinuria consistent with glomerular diseases.
PubMedID- 23898345 In glomerular diseases associated with proteinuria, tubular cells may be activated by elevated levels of normal and novel urinary proteins.
PubMedID- 24719498 Regardless of the stimuli (via local podocyte or systemic), pathological integrin signaling in podocytes might be considered a key event in the induction of glomerular diseases associated with proteinuria .
PubMedID- 24625990 glomerular disease with proteinuria may develop due to ischemia and results in a compensatory increase in the renal blood flow and glomerular filtration rate; such hyperfiltration, combined with glomerular hypertrophy, probably contributes to glomerulosclerosis.
PubMedID- 25246341 Conclusion: hqh granules have certain effect on delaying the development of primary glomerular disease with mild proteinuria and hematuresis in patients.
PubMedID- 24206430 Our data indicate that abatacept may stabilize beta1-integrin activation in podocytes and reduce proteinuria in patients with b7-1-positive glomerular disease.

Page: 1