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PedAM

Pediatric Disease Annotations & Medicines




Disease neuropathy
Symptom C0004093|weakness
Sentences 26
PubMedID- 23629918 Motor unit loss and weakness in association with diabetic neuropathy in humans.
PubMedID- 20665518 Igm mgus anti-mag neuropathy with predominant muscle weakness and extensive muscle atrophy.
PubMedID- 22203837 Additionally, mutations in mfn2 cause charcot-marie-tooth (cmt) disease-type 2a, the most common form of cmt disease and an inherited neuropathy leading to progressive weakness and sensory loss .
PubMedID- 26527893 Additionally, symptoms of symmetric significant distal weakness with polyneuropathy would not be expected to be encountered secondary to common causes of neuropathy such as glucose dysregulation or idiopathic axonal polyneuropathies, which are characteristically sensory predominant.
PubMedID- 24708634 The neuropathy typically presents with distal weakness and paresthesias, showing a symmetric stocking-glove distribution.
PubMedID- 22294875 The disease neuropathy with muscle weakness, ataxia, and retinitis pigmentosa (narp) is caused by t8993g heteroplasmic mtdna mutations.
PubMedID- 26434167 One case had severe peripheral neuropathy with muscle weakness, atrophy in left arm, and wasting on left hand.
PubMedID- 23526479 The classic ains is a pure motor neuropathy with weakness of flexor pollicis longus, flexor digitorum profundus to the 2nd and 3rd digits, and pronator quadratus muscles without sensory involvement 2.
PubMedID- 23098317 During icu treatment, a large proportion of the critically ill, mechanicallyventilated icu patients develop severe muscle wasting and weakness of limb musclesdue to acquired myopathy, neuropathy or a combination of both.
PubMedID- 22654708 It is a progressive sensorimotor neuropathy with distal weakness, areflexia, and reduced sensation in the extremities associated with acral mutilation.
PubMedID- 20736187 One patient exhibited a sensorimotor peripheral neuropathy with associated weakness of distal muscle groups in the upper and lower limbs, including weakness of thumb and finger abduction, ankle dorsiflexion and flexion and extension of the toes, all exhibiting an mrc grade 4.table 1clinical details for the 18 patients with acquired neuromyotoniapatientsage (years) sexfasciculations/ neuromyotoniacns features (mood disturbance, insomnia and confusion)anti-vgkc antibodiesautoimmune accompaniments140mpresentyesnegativeyes274mpresentyesnegativeyes371mpresentnonegativeno458mpresentyesnegativeyes517mpresentyespositiveno635mpresentyesnegativeyes743fpresentnonegativeno859mpresentyesnegativeyes958mpresentnonegativeyes1061fpresentnonegativeno1138mpresentyesnegativeyes1252mpresentnonegativeno1351fpresentnonegativeno1423mpresentnopositiveyes1547fpresentyespositiveyes1653fpresentnopositiveno1756mpresentnopositiveno1855mpresentyespositivenomean (sem)50 (5.6)fasciculation potentials and neuromyotonia were evident in all patients with acquired neuromyotonia.
PubMedID- 25473575 Nearly all css patients have been reported to show long-lasting and steroid-resistant neuralgia together with muscle weakness due to peripheral neuropathy 3.
PubMedID- 21687623 The most frequent clinical manifestation of acute attacks include vomiting, hypertension and tachycardia , and peripheral neuropathy with muscle weakness (42%–68%).
PubMedID- 22361624 Objectives: conduction block (cb) from focal neuropathy is often associated with weakness and fatigue in affected muscles.
PubMedID- 24084719 Dm is a multi-systemic disease and it is characterized by a generalized muscle weakness and wasting, associated with peripheral neuropathy, heart rhythm defects, and cataracts.
PubMedID- 24216706 Anti-hu antibodies have been reported in a mcc patient with painless proximal muscle weakness and in a patient with progressive sensorimotor neuropathy, autonomic neuropathy with gastroparesis, and encephalopathy with cognitive decline .
PubMedID- 26218529 The majority of patients showed the typical clinical picture of multifocal motor neuropathy with muscle weakness beginning at the hand muscles, spreading to more proximal muscles and to the lower limbs, and muscle atrophy during the course of disease.
PubMedID- 26197476 Monospecific anti-gt1a antibodies are associated with pharyngeal-cervical-brachial weakness, a localized subtype of acute motor axonal neuropathy, an axonal variant of gbs .
PubMedID- 22379454 Clinically, gbs manifests as an acute peripheral neuropathy with symmetric weakness reaching a peak by 4 weeks from onset, hyporeflexia or areflexia, and cytoalbuminemic dissociation in the cerebrospinal fluid (csf) with an elevated protein content and normal cell count 5.
PubMedID- 26467421 Symptoms of large fiber neuropathy include weakness of limbs and a loss of proprioception resulting in sensory ataxia, which are evaluated using conventional nerve conduction studies (ncs).
PubMedID- 20333310 It was reported that patients experienced significant sensory symptoms including numbness and tingling as well as limb weakness consistent with polyradiculoneuropathy, and was initially referred to as progressive inflammatory neuropathy (pin)1 now described as sensory predominant, immune-mediated polyradiculoneuropathy (ip).
PubMedID- 23400500 The final 15 % of clinicians presented with fasciculations associated with sensory symptoms or muscle weakness and were diagnosed with neuropathy (10 %) and als (5 %).
PubMedID- 24708773 Cancer treatments such as vinca-alkaloids may cause peripheral motor or sensory neuropathy, leading to clumsiness, muscular weakness and deficient motor skills.
PubMedID- 20083254 Hereditary sensory ataxic neuropathy associated with proximal muscle weakness in the lower extremities.
PubMedID- 20301352 Narp is characterized by proximal neurogenic muscle weakness with sensory neuropathy, ataxia, and pigmentary retinopathy.
PubMedID- 21465314 One case of grade 4 peripheral motor and sensory neuropathy associated with general weakness and pain was observed during treatment cycle 4 and possibly contributed to the death of the patient.

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