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PedAM

Pediatric Disease Annotations & Medicines




Disease influenza
Symptom C0555970|nasal infection
Sentences 19
PubMedID- 24064670 Following intranasal infection with influenza virus, the proportion of protected lung cd4 t cells increased dramatically, comprising >25% of total lung cd4 t cells as early as 3 days post-infection, peaking at 60–70% of lung cd4 t cells at day 10 p.i., and remaining elevated for up to 3 weeks p.i., with concomitant reductions in the proportion of labeled cd4 t cells at these timepoints (figure 1a and b).
PubMedID- 24675692 Interestingly, intranasal infection of mice infected with influenza a virus pr8/a/34 virus had little impact on the survival of irf-1−/− mice (figure s1), indicating a specific role of irf-1 in neurotropic vsv infections.
PubMedID- 22108420 Results: mice presented a mild syndrome after intranasal infection with a/guangzhou/333/99 (h9n2) influenza virus.
PubMedID- 20390297 Intranasal infection of mice with the influenza a hkx31 (h3n2) leads to transient weight loss, lung pathology, and high levels of pro-inflammatory cytokines in the bronchoalveolar lavage and plasma.
PubMedID- 21442033 Most inbred laboratory mice are highly susceptible to disease and death following intranasal infection with certain influenza viruses, including pr8 and wsn; in contrast, wild mice are resistant to even very high doses of the same viral strains .
PubMedID- 23486585 The lung vessels of male c57bl/6 mice were studied by immunohistochemical and stereometric methods on days 1, 3, 6, and 10 after intranasal infection with influenza a/h5n1 a/goose/krasnoozerskoye/627/05 virus.
PubMedID- 23168705 The inflammatory response in the upper respiratory tract after intranasal infection with influenza virus in ferrets showed an increase of neutrophils one day after infection.
PubMedID- 22615569 Upon intranasal infection with influenza a virus we again observed a sex bias in the viral load, measured in the lungs at day 8 post infection (figure 4a).
PubMedID- 21986509 The yogurt (0.4 ml/day) and eps (20 mug/day) were orally administered to balb/c mice for 21 days prior to intranasal infection with influenza virus a/pr/8/34 (h1n1).
PubMedID- 23405246 Following the generation of the mixed bone marrow chimeras, mice were rested for 3 months, before intranasal infection with influenza x31 (the parental virus from which the x31-ova variant was derived ).
PubMedID- 23979178 Senescence-accelerated mice were given het in drinking water for 2 weeks, followed by intranasal infection with influenza a virus strain pr8.
PubMedID- 22536437 In normal volunteers, intranasal infection with influenza a virus results in the release of tnf-α followed rapidly by the release of il-6 and other pro-inflammatory cytokines.
PubMedID- 21593156 Virus replication and pulmonary disease pathogenesis in ferrets following intranasal infection with a pandemic influenza virus strain (a/california/4/09 ca09), a human seasonal influenza h1n1 virus isolate (a/new caledonia/20/99 ncal99), a classical swine influenza h1n1 virus isolate (a/swine/iowa/15/30 sw30), or an avian h1n1 virus isolate (a/mallard/mn/a108-2355/08 mal08) were compared.
PubMedID- 25671696 Intranasal infections with influenza a/hkx31 (h3n2), a/pr8 (h1n1) and pandemic a/california/09 (h1n1) were used to generate influenza virus-specific memory t cells in mice as previously described .
PubMedID- 21896679 Mice lacking a functional nadph oxidase (cybb-deficient mice) can no longer produce ros, and intranasal infection with influenza virus was shown to increase the production of proinflammatory cytokines and promote cellular infiltration into the lungs (35, 36).
PubMedID- 20601181 At intranasal infection with influenza virus pr8 (a mouse-adapted h1n1 strain) after s-pt84 treatment, the survival rates of mice improved in a dose-dependent manner, and the titer of influenza virus in bronchoalveolar lavage fluids (balf) was significantly decreased by s-pt84 administration.
PubMedID- 25419628 Upon intranasal infection with influenza virus, setdb2gt/gt mice induced more cxcl1 compared to wt controls (fig.
PubMedID- 22783250 Increases in the number of lung nh cells were also observed upon intranasal infection with h1n1 influenza a virus (monticelli et al., 2011).
PubMedID- 23272202 We recently showed that intranasal infection of mice with influenza virus induced the accumulation of nk cells in the airways.

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