PedAM
Pediatric Disease Annotations & Medicines
| Disease | hiv infections |
| Symptom | C0004153|atherosclerosis |
| Sentences | 11 |
| PubMedID- 21345230 | An ongoing prospective study will aim at addressing the possible association of proinflammatory hdl with other markers of inflammation, immune activation and progression of atherosclerosis in patients with hiv infection. |
| PubMedID- 25609975 | Soluble markers such as scd163 and scd14, and the proportion of activated subsets of monocytes (cd14+/cd16+) and cd8 t-cells (hla-dr+cd38+), are increased in hiv patients.56–60,123,124 our group has recently reported that hiv patients also have lower levels of soluble tumor necrosis factor-like weak inducer of apoptosis (stweak), a multifunctional cytokine involved in various atherogenic processes through its interaction with fn14 and cd163.124 scd163 levels have been associated with the presence of subclinical atherosclerosis in patients with hiv infection. |
| PubMedID- 22192157 | Many studies have shown a higher rate of premature atherosclerosis in patients with hiv infection, leading to coronary, cerebrovascular, or peripheral arterial disease. |
| PubMedID- 22634976 | Aim: hiv infection is strongly associated with accelerated vascular atherosclerosis and increased cardiovascular events. |
| PubMedID- 26520571 | , epicardial adipose tissue was strongly associated with coronary artery calcium (an established marker of atherosclerosis) in patients with hiv infection on haart. |
| PubMedID- 25866592 | The inflammatory and prothrombotic changes caused by hiv infection may lead to atherosclerosis and subsequent plaque rupture. |
| PubMedID- 21273145 | Moreover, hiv infection is associated with accelerated coronary atherosclerosis and vasculopathy, although the mechanisms underlying these findings have not been determined. |
| PubMedID- 23079800 | Conclusion: atherosclerosis in hiv infection results from the interaction of multiple factors: an inflammatory and hiv-driven disease could prevail in the absence of therapy; metabolic, non-inflammatory causes may be more important in patients undergoing therapy. |
| PubMedID- 25699995 | It is well established that hiv infection is associated with premature atherosclerosis likely due to multiple factors including hiv‐related inflammation, hiv treatment‐related lipodystrophy and hypertension, and immunodeficiency.7–8 traditional risk factors such as lipids and smoking appear to be more important than hiv disease‐related variables in producing increased cimt in hiv‐infected adults.9 to our knowledge, echogenicity of the carotid artery intima‐media complex has not been studied in an hiv‐infected population. |
| PubMedID- 26169283 | An accumulating body of evidence suggests that hiv infection leads to accelerated atherosclerosis . |
| PubMedID- 21646832 | This case highlights the association between immunosuppression with haart, particularly protease inhibitors, and the development of accelerated atherosclerosis in patients with hiv infection. |
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