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PedAM

Pediatric Disease Annotations & Medicines




Disease glucose intolerance
Symptom C0011847|diabetes
Sentences 38
PubMedID- 20879973 We aimed to systematically identify and evaluate those studies that have investigated the impact of lifestyle interventions on the prevention of the development of incident type 2 diabetes in those with glucose intolerance.
PubMedID- 21494686 In rodents, models of gestational diabetes produce offspring with glucose intolerance, impaired insulin secretion, insulin resistance and increased adiposity , , .
PubMedID- 21818460 Combination of weight loss and exercise to reduce the incidence of type 2 diabetes in patients with glucose intolerance should not be dismissed .
PubMedID- 26324089 In spite of glucose intolerance patients with gck-diabetes showed normal incretin effect, whereas patients with hnf1a-diabetes showed an impaired incretin effect.
PubMedID- 26347815 In addition to being obese, the ob/ob mice are reported to exhibit hyperphagia, a transient diabetes-like syndrome of hyperglycemia, glucose intolerance, elevated plasma insulin, subfertility, impaired wound healing, and increased hormone production from pituitary and adrenal glands, and apparently they are hypometabolic and hypothermic .
PubMedID- 24518493 However, in contrast to type 2 diabetes, onset and progression of glucose intolerance in pancreatic cancer–induced diabetes occur paradoxically in the face of ongoing, often profound, weight loss.
PubMedID- 24899904 Left ventricular changes are frequently reported in diabetes in existence of glucose intolerance .
PubMedID- 20862330 Dysregulated branched-chain metabolism may make an independent contribution to development of insulin resistance and glucose intolerance, ultimately leading to type 2 diabetes .
PubMedID- 23928458 For instance, short sleep duration (<7 hours of sleep per night) and poor sleep quality are associated with cardiovascular morbidity and metabolic disorders such as glucose intolerance, which may lead to obesity, diabetes, heart disease, and hypertension (1).
PubMedID- 21605417 Both models, b6.cg-m+/+ lepob/j or ob/ob and the b6.v-lepdb/j or db/db), exhibit obesity, hyperphagia, a diabetes-like syndrome of hyperglycemia, glucose intolerance, elevated plasma insulin, and subfertility .
PubMedID- 23573479 Male kk/ta mice spontaneously exhibit type 2 diabetes associated with hyperglycemia, glucose intolerance, hyperinsulinemia, mild obesity and microalbuminuria, conditions which are more severe than those found in the female (2-4).
PubMedID- 22936178 Downregulation of glut4 expression in skm is a major mechanism, which contributes to glucose intolerance associated with type 2 diabetes (31).
PubMedID- 21243857 The rate of occurrence of variable glucose intolerance was 28% with frank diabetes mellitus diagnosed in 9% of the cases of hyperthyroidism.
PubMedID- 23298663 Aims: to assess and compare costs associated with diabetes and lesser degrees of glucose intolerance in australia.
PubMedID- 25898207 Insulin resistance and islet β-cell dysfunction are important in the pathogenesis of glucose intolerance leading to type 2 diabetes .
PubMedID- 22391950 Several randomised controlled trials have demonstrated that moderate changes in lifestyle including both dietary behaviour and physical activity clearly decrease the risk of progression to diabetes in humans with glucose intolerance.
PubMedID- 22974102 Conclusions: alcohol abstinence might influence diabetes-related factors of alcohol-dependent patients with glucose intolerance.
PubMedID- 23822149 High admission glucose in patients with diabetes is mainly due to glucose intolerance in the setting of diabetes.
PubMedID- 21257329 Exposure in utero to maternal diabetes leads to glucose intolerance and high blood pressure with no major effects on lipid metabolism.
PubMedID- 23983688 Gestational diabetes is a diagnosis of any glucose intolerance detected in pregnancy.
PubMedID- 24719845 Risk factors for diabetes include previous history of impaired glucose intolerance; high-risk ethnic groups (i.e., asian, african, and hispanic); positive family history or gestational diabetes; age greater than or equal to 45 with either a body mass index (bmi) greater or equal to 25 or presence of hypertension; and established cardiovascular risk factors or disease.
PubMedID- 24836607 In subjects without hiv and without diabetes, the presence of glucose intolerance (ifg or impaired glucose tolerance) is associated with a significant increase in c-imt .
PubMedID- 21926287 Objective the increased risk of type 2 diabetes in women with glucose intolerance in pregnancy is mediated by deterioration of their beta-cell function, which occurs as early as the first year postpartum.
PubMedID- 23998129 Overall, the preexisting maternal diabetes leads to glucose intolerance, insulin resistance, and impaired insulin sensitivity and beta -cell function in the offspring at different postnatal periods.
PubMedID- 19788432 In this review, we discuss current recommendations for managing women with gdm in the postnatal period, with particular attention to postpartum diabetes screening, prevention of future glucose intolerance and family planning.
PubMedID- 24053606 Since diabetes is a disease of glucose intolerance, most studies have focused on elucidating the role of hyperglycaemia on cv complications.
PubMedID- 23878520 5 assessed both lifestyle intervention and metformin against placebo intervention in the prevention of type 2 diabetes in individuals with impaired glucose intolerance.
PubMedID- 24069200 Since gestational diabetes is a form of glucose intolerance, it is critical that women with gdm acquire knowledge about foods that contain carbohydrate to facilitate food choices that will not result in hyperglycemia.
PubMedID- 21750276 Women with gdm who develop diabetes face longer periods of glucose intolerance than other glucose-intolerant adults (12), and earlier diagnosis and treatment might be advantageous.
PubMedID- 21194350 We also observed that reduced synthesis of these selenoproteins caused by overexpression of an i(6)a(-) mutant selenocysteine trna promotes glucose intolerance and leads to a diabetes-like phenotype.
PubMedID- 21357466 In summary, increased dosage of δ40p53 in mice promotes hypoinsulinemia and glucose intolerance, ultimately leading to overt diabetes with age and early death.
PubMedID- 25977929 Prepregnancy obesity (30.0+), parity, maternal height, excess pregnancy weight gain, and glucose intolerance without gestational diabetes were significantly associated with a higher infant weight/length ratio in this model.
PubMedID- 23811337 The kkay mice represent a model for obesity and type 2 diabetes, with spontaneous development of hyperglycemia, hyperinsulinemia, glucose intolerance, and obesity .
PubMedID- 23570346 In previous studies we showed that in japanese-brazilians, low plasma level of total adiponectin is an independent predictor of glucose intolerance and subjects with type 2 diabetes compared with normal glucose tolerant subjects have reduced levels of total adiponectin .
PubMedID- 20304061 Results from small underpowered trials and post-hoc analyses of data from larger trials designed for bone-specific outcomes show no effect of vitamin d supplementation on glycemia in healthy adults but vitamin d may retard the progression to diabetes in adults with glucose intolerance.
PubMedID- 23634949 Recognizing and treating diabetes or any degree of glucose intolerance in pregnancy results in lowering maternal and fetal complications.
PubMedID- 24186867 Although population studies and experimental models indicate that maternal obesity and diabetes increase risk of obesity and glucose intolerance in offspring, the mechanisms underlying such associations remain unclear.
PubMedID- 23319937 Thus, l-pgds is an important mediator of muscle and adipose glucose transport which is modulated by glycemic conditions and plays a significant role in the glucose intolerance associated with type 2 diabetes .

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