Disease | hepatitis |
Symptom | |chronic infection |
Sentences | 332 |
PubMedID- 24168254 | From 2002 to 2011, the standard of care treatment for chronic infection with hepatitis c virus (hcv) was 24 or 48 weeks of therapy with pegylated interferon-alfa (peg-ifn) and ribavirin (rbv). |
PubMedID- 21569429 | Indeed, the main risk factors for the development of hcc among the chinese population are the presence of liver cirrhosis and chronic infection with hepatitis b virus 4. |
PubMedID- 23010773 | Introduction: adverse effects appearing during combined peg-interferon and ribavirin antiviral treatment against chronic infection with the hepatitis c virus are a major cause for treatment failures and abrupt interruption. |
PubMedID- 25820703 | Background: chronic infection with hepatitis c virus (hcv) genotype 2 or 3 can be treated with sofosbuvir without interferon. |
PubMedID- 22514698 | The important risk factors are liver fluke infection and possible involvement from chronic infection with hepatitis b and c viruses . |
PubMedID- 23634229 | The etiological importance of chronic infection with the hepatitis b virus (hbv) and the hepatitis c virus (hcv) in hcc has been well established 3. |
PubMedID- 21813377 | Morbidity due to chronic infection with chronic-hepatitis-b (hbv) and hcv viruses has become a leading cause of death in hiv-infected patients with synchronous chronic viral hepatitis. |
PubMedID- 21605699 | For example, chronic infections with hepatitis b or hepatitis c virus are well described etiological factors for the generation of liver inflammation and hcc . |
PubMedID- 25713800 | chronic infection with hepatitis b virus (hbv) is the leading cause of liver cirrhosis and hepatocellular carcinoma worldwide. |
PubMedID- 21994707 | chronic infection with hepatitis c virus (hcv) frequently leads to liver cirrhosis or liver cancer. |
PubMedID- 22187946 | chronic infection with hepatitis c virus (hcv), mainly genotype 1, has been shown to be associated with insulin resistance and type 2 diabetes. |
PubMedID- 26219465 | There is consistent evidence that the burden of chronic liver disease is greater among certain ethnic groups, particularly in areas characterized by high rates of chronic infection with hepatitis viruses 9. |
PubMedID- 25927412 | Major risk factors for development of hcc are chronic infection with hepatitis b virus (hbv) or hepatitis c virus (hcv), liver cirrhosis, habitual alcohol abuse, high cigarette smoking, and exposure to aflatoxin b1 . |
PubMedID- 25879744 | In these high-incidence populations, except for japan, chronic infection with hepatitis b virus (hbv) and aflatoxin exposure were recognized as major risk factors for plc. |
PubMedID- 25937322 | chronic infection with hepatitis b virus (hbv) occurs commonly and complications that arise from persistence of the virus are associated with high mortality. |
PubMedID- 23419149 | China and africa are areas of high hcc incidence where the primary cause of hcc is chronic infections with the hepatitis b virus (hbv), and dietary aflatoxin exposure being an important cofactor . |
PubMedID- 22437530 | Hcc is a major public health issue in korea due to its high incidence related to chronic infection with hepatitis b and c virus . |
PubMedID- 26357633 | chronic infection with hepatitis c virus (hcv) carries a significant global health burden in both children and adults, with approximately 3% of the world's population infected. |
PubMedID- 21760886 | chronic infection with hepatitis c virus (hcv) is an important cause of liver cancer and end-stage liver disease in the united states and worldwide . |
PubMedID- 20331884 | chronic infections with hepatitis b virus (hbv) and hepatitis c virus (hcv) are a leading cause of hepatocellular carcinoma, a condition whose incidence rate remains high in the developing world and is rising across most developed countries 1. |
PubMedID- 23151512 | chronic infection with hepatitis c virus (hcv) is an important cause of end stage liver disease worldwide. |
PubMedID- 23617810 | chronic infection with hepatitis c virus (hcv) affects 130–200 million people worldwide 1. |
PubMedID- 25034398 | Background: chronic infection with hepatitis b virus (hbv) is the major risk factor of hepatocellular carcinoma (hcc). |
PubMedID- 23613990 | chronic infection with hepatitis c virus (hcv) leads to exhaustion and death of hcv-specific t-cells (reviewed in ) but may also cause defects in overall immune function. |
PubMedID- PMC3882569 | chronic infection with hepatitis c virus (hcv) induces a chronic stimulation of b lymphocytes and, in some cases, this stimulation can lead to chronic lymphoid disorders like non-hodgkin lymphoma. |
PubMedID- 22611419 | In chronic infection with hepatitis b virus or hcv, specific ctls are few and engage few specific targets; they also display anergic characteristics with reduced type 1 cytokine secretion . |
PubMedID- 25615823 | In spite of a number of well-established risk factors for liver cancer, including chronic infection with hepatitis b virus (hbv) or hepatitis c (hcv), less attention, however, has been paid to the role of dietary factors except for alcohol abuse and aflatoxin contamination in the development of this malignancy . |
PubMedID- 23437305 | In asia, people are at higher risk of developing hcc because of chronic infection with hepatitis b virus (hbv) –. |
PubMedID- 25969457 | chronic infection with hepatitis b virus, hepatitis c virus (hcv) and alcohol consumption are the leading causes of cirrhosis worldwide. |
PubMedID- 21407274 | The prevalence of chronic infection with the hepatitis c virus (hcv) in patients with chronic kidney disease is higher than in the general population. |
PubMedID- 22448225 | Background and aims: chronic infection with the hepatitis b virus (hbv) is a major health issue worldwide. |
PubMedID- 22158321 | In addition to these associations, there is substantial evidence for a causativerelationship between chronic infection with hepatitis c virus (hcv) and non-hodgkinlymphoma (nhl), an association that has been the subject of several recentsystematic reviews (gisbert et al, 2003;matsuo et al, 2004; dal maso and franceschi, 2006). |