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PedAM

Pediatric Disease Annotations & Medicines




Disease myocardial infarction
Symptom |heart failure
Sentences 128
PubMedID- 22873828 Inclusion of a supervised outpatient cardiac rehabilitation program is clearly more cost-effective than not including cardiac rehabilitation program (“usual or standard care”) into the overall management of patients after myocardial infarction or those with chronic heart failure.
PubMedID- 24456675 Background: adverse remodeling of the left ventricle (lv) following myocardial infarction (mi) leads to heart failure.
PubMedID- 24112470 The results were essentially unchanged when subjects with incident myocardial infarction or hospitalizations because of heart failure were censored from the analysis (hr 1.30, 95% ci 1.13-1.51; p for trend = 0.001).
PubMedID- 26209495 Results: patients having cuf were more likely to have diabetes, vascular disease, chronic obstructive pulmonary disease, congestive heart failure, history of a myocardial infarction, or an intraaortic balloon pump (p < 0.05).
PubMedID- 22919539 Aldosterone increases angiotensin-converting enzyme and at1 receptor in the paraventricular nucleus (pvn) of the hypothalamus in chronic heart failure with postmyocardial infarction .
PubMedID- 23133676 Based on our results we assume that the reported increased mortality is rather a consequence of the concomitant heart disease than the conduction delay in itself as β-ar blockade basically shows positive effects on mortality in patients after myocardial infarction or in patients with congestive heart failure , .
PubMedID- 21375762 The expectation that bp agents have beneficial effects independent of the blood pressure-lowering one (pleiotropic effects) is still deep-rooted in physician's minds, but it has been repeatedly dismissed by independent clinical trials and by a large meta-analysis .
PubMedID- 20874717 (a–c) toluidine blue-positive mast cells are shown (as indicated by black arrowheads) in the endocardium (upper panels) of sham and post-myocardial infarction (mi) rats with heart failure treated with tat or βiiv5-3.
PubMedID- 24072687 On the contrary, only 19.3 % of respondents inquired regularly or often about pde-5 inhibitor use, while 43.6 % said they informed patients about the possibility to use a pde-5 inhibitor after a myocardial infarction (mi) or in patients with heart failure.
PubMedID- 21147287 Primary percutaneous coronary intervention (pci) decreases myocardial damage and reduces the occurrence of mechanical complications and acute heart failure in patients with st elevation myocardial infarction (stemi).
PubMedID- 25684642 Do statins reduce the risk of myocardial infarction in patients with heart failure.
PubMedID- 22106414 Aims: myocardial infarction leads to heart failure and death.
PubMedID- 22911790 Background: activation of the renin-angiotensin-system (ras) plays a key pathophysiological role in heart failure in patients with hypertension and myocardial infarction.
PubMedID- 26424212 Serum uric acid is associated with mortality and heart failure hospitalizations in patients with complicated myocardial infarction: findings from the high-risk myocardial infarction database initiative.
PubMedID- 22388688 Heart transplantation is one of the only treatment options for heart failure due to an acute myocardial infarction, but limited donor supply and organ rejection limit its widespread use.
PubMedID- 20407864 The effects of electric currents applied during absolute refractory period (arp) on the cardiac function of rabbits with heart failure due to myocardial infarction (mi), and the safety of this method were investigated.
PubMedID- 26263161 Background and purpose: myocardial infarction leads to heart failure.
PubMedID- 21517671 A novel radiological score to assess lung fluid content during evolving acute heart failure in the course of acute myocardial infarction.
PubMedID- 22474044 Postmortem examination revealed coronary arteriosclerosis and myocardial infarction leading to congestive heart failure.
PubMedID- 24339455 Chronic heart failure, as a result of acute myocardial infarction, is a leading cause of death worldwide.
PubMedID- 24839602 In particular, anakinra improved left ventricular remodelling and numerically lowered the incidence of heart failure in patients with acute myocardial infarction and st-segment elevation.
PubMedID- 21969217 Upright t waves in lead avr are associated with cardiac death or hospitalization for heart failure in patients with a prior myocardial infarction.
PubMedID- 25522363 As heart failure due to myocardial infarction remains a leading cause of morbidity worldwide, cell-based cardiac regenerative therapy using cardiac progenitor cells (cpcs) could provide a potential treatment for the repair of injured myocardium.
PubMedID- 20201885 myocardial infarction can lead to irreversible heart failure.
PubMedID- 22453289 The presence of diabetes substantially accelerates development of heart failure in patients with myocardial infarction , hypertension , leading to poorer prognosis.
PubMedID- 24751462 Objectives: to evaluate the efficacy of a short-term intravenous infusion of levosimendan in patients with heart failure due to acute myocardial infarction (ami).
PubMedID- 20730062 A meta-analysis of all randomized trials showed that amiodarone reduced total mortality by 10 to 19%.30 the risk reduction was similar in primary prevention after myocardial infarction or in patients with congestive heart failure (chf), and in secondary prevention after cardiac arrest.30 in a pooled database from 2 similar randomized clinical trials (the european amiodarone myocardial infarction trial (emiat) and the canadian amiodarone myocardial infarction trial (camiat)), that evaluated use of amiodarone in primary prevention in patients recovering from myocardial infarction, cardiac death and arrhythmic death or resuscitated cardiac arrest, were significantly lower in patients receiving amiodarone, compared to placebo (p = 0.05 and 0.03, respectively), if they were also receiving beta-blockers.31 there appeared to be no benefit of amiodarone over placebo in patients not receiving beta-blockers.
PubMedID- 22132230 Structural remodeling of the left ventricle, which is initiated by pathological events such as hypertension or myocardial infarction, can ultimately lead to heart failure (hf).

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