Disease | septicemia |
Symptom | |pneumonia |
Sentences | 67 |
PubMedID- 25530550 | Antithrombin and mortality in severe pneumonia patients with sepsis-associated disseminated intravascular coagulation: an observational nationwide study: reply. |
PubMedID- 21067543 | Another good example is streptococcus pneumoniae, a major cause of sepsis, pneumonia, and meningitis . |
PubMedID- 25611174 | Antithrombin or thrombomodulin administration in severe pneumonia patients with sepsis and disseminated intravascular coagulation: comment on two papers. |
PubMedID- 26576275 | On the thirteenth day after the administration of the chemotherapy the patient developed severe cmv- and e.coli pneumonia with consecutive sepsis and acute kidney transplant failure requiring transmission to an intensive care unit and invasive ventilation. |
PubMedID- 23227074 | In this case, the patient presented sepsis with pneumonia and had signs of septic embolism with chest imaging such as poorly defined nodules in the lung periphery. |
PubMedID- 23709686 | pneumonia is frequently associated with sepsis, characterized by a nonresolving hyperinflammation. |
PubMedID- 23480440 | There is controversy about the potential use of srage as a biomarker of sepsis in patients with pneumonia, however. |
PubMedID- 21779281 | The causes of non-cardiac-related deaths were sepsis associated with pneumonia and upper gastrointestinal bleeding (table 3). |
PubMedID- 22606398 | Filgrastim, a granulocyte colony-stimulating factor (g-csf) has been used as adjuvant therapy in patients of pneumonia with sepsis 3. |
PubMedID- 26005330 | There exist several difficulties and consequences in conventional therapies as: 1) time is crucial in resuscitation of septic patients; in delayed treatments, patients may experience permanent organ failure; for example, in lung infection (pneumonia) with sepsis, patients may need respiratory support or septic shock related to a limb infection patient may end up to limb amputation;25 2) in improper antibiotic therapies, each patient is likely to have the potential for complications related to invasion of bacterial antigens and their toxins (bats) to internal organs, which may lead to organ dysfunction, damage, or death; however, death is usually due to multiorgan dysfunction (liver, kidney, or lung failure);17,26,27 3) patients are with overwhelmed immune response to infection;28 4) there exist complications in patients who are at greater risk for infection such as people with diabetes,29 cancer, suppressed immune systems, and elderly patients;14,30 and 5) there is spread of antibiotic resistance and the appearance of multiple antibiotic-resistant pathogenic bacteria, such as mrsa, which is an increasingly prevalent problem that complicates the care of many patients.30,31 however, several more can be added to the above. |
PubMedID- 23420572 | The patient succumbed to healthcare-acquired pneumonia with severe sepsis due to pseudomonas aeruginosa, 10 days after admission. |
PubMedID- 22184529 | In our second case, no venous access device had been in place during the onset of fever, and preceding bacterial sepsis with pneumonia might have caused pulmonary tissue damage, facilitating aspergillus entry along with the use of broad spectrum antibiotics. |
PubMedID- PMC3363827 | The primary reason for admission was sepsis (61%), with pneumonia the commonest source (27%) and 42% admitted with neutropenic sepsis. |
PubMedID- 24475220 | Methods: a total of 196 patients with pneumonia-induced sepsis and 213 age- and sex-matched healthy volunteers participated in our study from july 2012 to july 2013 in guangzhou, china. |
PubMedID- 25493910 | One patient died after developing nosocomial pneumonia with severe sepsis after debridement. |
PubMedID- 21502443 | Costs were highest when sepsis occurred with pneumonia or failure to wean the patient from mechanical ventilation after 48 hours. |
PubMedID- 23481491 | The pretreatment of mice subjected to pneumonia-induced sepsis with zndpbg (zinc deuteroporphyrin 2,4-bis glycol), a nonspecific ho inhibitor, increased the number of neutrophils in the bronchoalveolar spaces, reduced the bacterial load at the site of infection, and prevented the upregulation of cd11b and the downregulation of cxcr2 on blood neutrophils. |
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