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PedAM

Pediatric Disease Annotations & Medicines




Disease asthma
Symptom |infections
Sentences 68
PubMedID- 25525736 Such studies indicate the immunological mechanisms that may underpin the association between early-life respiratory infections and the development of more severe asthma and may facilitate the development of tailored preventions and treatments.
PubMedID- 21448631 We conclude that not all viral infections in children with asthma lead to an asthma exacerbation and the attributing effect of different triggers of asthma exacerbations in children varies across different time periods and across different localities.
PubMedID- 21996340 Viral respiratory tract infections are strongly associated with asthma exacerbation, but the effects of respiratory viruses on airway epithelial barrier function are not well understood.
PubMedID- 22754709 In fact, nasal and bronchial diseases often coexist and upper respiratory tract infections (urti) are capable of exacerbating asthma,2 while rhinitis has been identified as an independent risk factor for asthma development.3 the upper and lower respiratory tracts form a continuum and share many anatomical and histological properties and an important feature: the passage of air into and out of the lungs.
PubMedID- 20528604 In stratified analyses, the increased risk of asthma in children with recurrent ear infections in infancy persisted among children whose parents reported no lifetime history of rhinitis but not in children who had a history of rhinitis.
PubMedID- 26137528 The results indicate that igg1 isotype antibody responses specific for the n-terminal fragments of the vp1 coat protein may serve as serologic markers for clinically relevant rv infections associated with asthma exacerbations that might be valuable in future epidemiologic studies (niespodziana et al., 2015--in press).
PubMedID- 21687760 These findings suggest that the recurrence of piv infections in patients with asthma may contribute to prolonged airway inflammation and airway structural changes typical of the disease (pelaia et al., 2006).
PubMedID- 25901797 It is important for clinicians to detect and treat bacterial infections in patients with asthma exacerbation in clinical practice.
PubMedID- 26289993 Hrv subtype c (hrv-c) infections were associated with asthma (p = 0·02).
PubMedID- 23098057 The immediate sign that the airways diseases coexist is given by the fact that upper respiratory tract infections are capable of exacerbating asthma, while rhinosinusitis has been identified as an independent risk factor for asthma development .
PubMedID- 25866598 Upper respiratory viral infections are also associated with 80-85% of asthma exacerbations in school-age children 2.
PubMedID- 23451114 Respiratory tract infections may lead to asthma, and our findings highlight the negative impact of these types of illnesses on the emotional functioning of pediatric patients.
PubMedID- 25191433 Considering the role of selectin in pulmonary infections leading to bronchial asthma and creation of airway hyper-reactivity (ahr), they evaluated the role of a protein nanoparticle p-selectin antagonist with anti-inflammatory effects in allergic airways disease in in-vitro asthmatic models (15).
PubMedID- 20410486 These cells may be particularly important in determining the nature of immune responses to viral infections in patients with allergic asthma as well those with other atopic diseases.
PubMedID- 26362307 We investigated early-life respiratory infections as predictors of the development of asthma in a 20-year prospective cohort study (the espoo cohort study, 1991-2011).
PubMedID- 20228193 Chlamydial respiratory infections are associated with asthma, but how these th1-inducing bacteria influence th2-mediated asthma remains unknown.
PubMedID- 26020408 Human rhinoviruses (hrv) cause respiratory infections and are associated with asthma development.
PubMedID- 23060458 Although rhinoviral infections, a major cause of asthma exacerbations, occur predominantly in upper airway bronchial epithelial cells, monocytic-lineage cells are implicated in establishing the inflammatory microenvironment observed during the disease.

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