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PedAM

Pediatric Disease Annotations & Medicines




Disease hepatitis b
Symptom |infection
Sentences 596
PubMedID- 22844019 Clinical care versus ethical obligations: hiv-1 and -2 co-infection with hepatitis b in a pregnant jehovah's witness.
PubMedID- 23712427 Demographic characteristics including age, sex, hcv risk factors, hcv genotypes, alcohol consumption, markers of chronic infection with the hepatitis b virus and hiv, hcv viral load, liver biopsy data, and hcv treatment were extracted from clinical databases.
PubMedID- PMC3112874 Co-infection with hepatitis b occurred in 16 subjects and hepatitis c in 11.
PubMedID- 24521641 Background & objectives: co-infection with hepatitis b virus (hbv) and hepatitis c virus (hcv) in human immunodeficiency virus (hiv) infected individuals results in increased hepatic complications.
PubMedID- 24325818 Acute hepatitis b patients with hiv infection have six times greater risk of developing chronic hepatitis b, with higher viral replication, rapid progression to end-stage liver disease and shorter survival 6.
PubMedID- 24351862 Hcc is etiologically unique in that it arises from various types of chronic hepatic diseases such as infection with hepatitis b or c virus (hbv, hcv), and alcohol abuse.
PubMedID- 25927412 Major risk factors for development of hcc are chronic infection with hepatitis b virus (hbv) or hepatitis c virus (hcv), liver cirrhosis, habitual alcohol abuse, high cigarette smoking, and exposure to aflatoxin b1 .
PubMedID- 22693513 Coinfection with hepatitis b or c was defined as serologic evidence of hepatitis b or c infection prior to or at the time of itp diagnosis.
PubMedID- 23907803 infection with hepatitis b virus (hbv) is the most common cause of liver disease worldwide.
PubMedID- 24254267 This cross-sectional study aims to investigate: i- the prevalence of hepatitis d virus co-infection in children with hepatitis b virus infection in western turkey; ii- the influence of neonatal hepatitis b virus vaccination on hepatitis d virus co-infection rate; and iii- the impact of co-infection on prognosis of liver disease.
PubMedID- 24708767 Exclusion criteria included co-infection with hepatitis b or human immunodeficiency virus, co-existent autoimmune or metabolic liver disease, active drug-induced hepatitis, decompensated cirrhosis, evidence of severe retinopathy, neoplastic disease, coronary artery or cerebrovascular disease, history of clinically relevant psychiatric disease.
PubMedID- 25750558 Chronic infection with the hepatitis b virus (hbv) is a major cause of cirrhosis and hepatocellular carcinoma (hcc) worldwide.
PubMedID- 21994862 A total of 188 patients were excluded from the trial because of concurrent infection with hepatitis b (38 patients), presence of hepatocellular carcinoma (8 patients), decompensated cirrhosis (11 patients), history of psychiatric troubles (18 patients), presence of baseline anaemia ((hb < 13 g/dl for men and <12 g/dl for women)—39 patients), low absolute neutrophil count (anc) <1500/mm3 (16 patients), and low baseline platelet count <75,000/mm3 (58 patients).
PubMedID- 23761295 Aflatoxin b1 in particular is a potent liver carcinogen, and hepatocellular carcinoma (hcc) risk is multiplicatively higher for individuals exposed to both aflatoxin and chronic infection with hepatitis b virus (hbv).
PubMedID- 25178323 Conclusions: infection with hepatitis b and c viruses induces inos expression in hepatocytes, suggesting that no overproduction might have an important role in progression of chronic viral hepatitis to cirrhosis.
PubMedID- 26069796 Six (6%) of the patients had co-infection with hepatitis b virus.
PubMedID- 22235211 Progressive fibrosis is the hallmark of an unfavorable course and is related to several factors such as male gender, contracting the disease at an older age, features of the metabolic syndrome, iron overload, ethanol abuse, and co-infection with hepatitis b or human acquired immunodeficiency viruses.
PubMedID- 23274669 Chronic infection with hepatitis b virus (hbv) is associated with impairment of t and nk cell immunity.
PubMedID- 21155156 infection with hepatitis b virus (hbv) is common in aboriginal australians, but current seroprevalences in northern territory communities are unknown.
PubMedID- 26155840 Chronic infection with hepatitis b (hbv) or hepatitis c virus (hcv) is considered the most important risk factor in hcc tumorigenesis 3.
PubMedID- 22200181 Exclusion criteria included co-infection with hepatitis b virus (hbv) and/or schistosome infection.
PubMedID- 23906236 A prominent factor in hepatocellular carcinoma (hcc) is chronic infection with hepatitis b virus (hbv) and hepatitis c virus (hcv).
PubMedID- 23230390 chronic inflammation and subsequent cirrhosis of the liver, most cases of which are induced by persistent infection with hepatitis b virus (hbv) or hepatitis c virus (hcv), contribute to the development of hcc.
PubMedID- 21151655 Major hcc risk factors include infection with hepatitis b (hbv) or c viruses (hcv) and cirrhosis associated with chronic inflammation.
PubMedID- 22085801 Basic socio-demographic characteristics, data on the clinical course (occurrence of opportunistic infections, death), co-infection with hepatitis b and c, tst, antiretroviral therapy, co-medication (prophylaxis and treatment of opportunistic infections), immunologic and virologic parameters are collected at enrolment into the study and every 6 months thereafter on standardised data collection forms.
PubMedID- 22742931 Major risk factors of hcc include infection with hepatitis b or c viruses, alcohol and non-alcoholic fatty liver disease.
PubMedID- 23098862 Chronic infection with the hepatitis b virus can lead to hepatocellular carcinoma and cirrhosis in up to 25% of infected individuals.
PubMedID- 23788462 Chronic infection with hepatitis b virus (hbv) can result in progressive liver disease, hepatocellular carcinoma (hcc), and death in as many as 25% of hepatitis b surface antigen (hbsag) carriers.1 in patients with end-stage liver disease, liver transplantation (lt) is often the only treatment option.
PubMedID- 20820946 The vast majority of cases are a consequence of a preexisting chronic viral infection due to either hepatitis b with or without associated hepatitis d or hepatitis c .
PubMedID- 22880052 The cause for the higher incidence rate of liver cancer among males compared to females is not completely understood, but may be partly explained by the sex-specific prevalence of risk factors like alcohol drinking, cigarette smoking and infection with hepatitis b virus (hbv) and hepatitis c virus (hcv) .
PubMedID- 22540175 For example, in sub-saharan africa, estimates for the risk of transfusion-associated infection with hiv, hepatitis b and hepatitis c are 1, 4.3 and 2.5 infections per 1,000 units of blood respectively .
PubMedID- 20824087 Persistent infection with hepatitis b virus (hbv) causes chronic hepatitis b which frequently progresses to hepatocellular carcinoma, a leading cause of cancer-mediated mortality worldwide.
PubMedID- 20600328 Establishment of an infection with hepatitis b virus (hbv) requires synthesis and maintenance of a covalently closed circular dna (cccdna) form of the viral genome in the nucleus of host cells.
PubMedID- 26357626 infection with hepatitis b virus (hbv) is a worldwide health problem.
PubMedID- 25959919 Hepatocellular carcinoma is the third leading cause of cancer death worldwide.1 several risk factors for hcc have been reported, including infection with hepatitis b and hepatitis c viruses, and alcohol consumption.
PubMedID- 20677332 The main etiology of hcc is chronic infection with hepatitis b and hepatitis c viruses.
PubMedID- 21841730 Reactivation of infection with hepatitis b virus (hbv) is a potentially serious complication of immunosuppression, which can be identified and efficiently prevented.
PubMedID- 22740812 Serological tests indicated no recent infection with hepatitis b, hepatitis c, human immunodeficiency virus or syphilis, while seropositive findings were noted for cmv-igm, cmv-igg and h. pylori antibodies.
PubMedID- 26469342 Exclusion criteria included evidence of alcoholic liver disease and co-infection with hepatitis b and hepatitis c. age, gender, height, weight, body mass index (bmi), serum aspartate aminotransferase (ast), alanine aminotransferase (alt), total bilirubin and platelet counts were recorded, and apri scores were calculated at the time of histopathological examination.
PubMedID- 24348636 It has been suggested that infection with hepatitis b and hepatitis c viruses may trigger immunologic intolerance to gluten (48, 49).
PubMedID- 25331321 Approximately one third of the world’s population has serological evidence of past or present infection with hepatitis b virus (hbv) 1.
PubMedID- 22536438 It has been suggested that infection with hepatitis b virus (hbv) or hepatitis c virus (hcv) increases the aberrant methylation of tumor suppressor genes in hcc, including the gstp1 gene , , , , , in hbv-infected hcc but not in non-hbv-infected hcc or in non-hcv-infected hcc.
PubMedID- 24203912 Hepatoprotectant use and infection with chronic hepatitis b were associated with increased risk of liver injury.
PubMedID- 23675384 infection with hepatitis b virus (hbv) may lead to acute, chronic and sometimes occult infection.
PubMedID- 23527043 patients were excluded if they had any of the following: any other coexistent liver disorders (alcoholic liver disease, autoimmune hepatitis, primary biliary cirrhosis, sclerosing cholangitis, wilson's disease and α1-antitrypsin deficiency); co-infection with hepatitis b or anti-human immunodeficiency virus; active use of illicit intravenous drugs; or a history of an uncontrolled psychiatric condition, pregnancy, decompensated cirrhosis or overt hepatic failure.
PubMedID- 21994607 The hepatitis delta virus (hdv) is the smallest known mammalian pathogen and is responsible for exacerbation of hepatitis disease progression upon co-infection with the hepatitis b virus (hbv).

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